記事
Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases
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すぐに読む
CiNii Research
Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases
- 資料種別
- 記事
- 著者
- Kendra D. Martynほか
- 出版者
- Elsevier BV
- 出版年
- 2006-01
- 資料形態
- デジタル
- 掲載誌名
- Cellular Signalling 18 1
- 掲載ページ
- p.69-82
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デジタル
- 資料種別
- 記事
- 出版年月日等
- 2006-01
- 出版年（W3CDTF）
- 2006-01
- タイトル（掲載誌）
- Cellular Signalling
- 巻号年月日等（掲載誌）
- 18 1
- 掲載巻
- 18
- 掲載号
- 1
- 掲載ページ
- 69-82
- 掲載年月日（W3CDTF）
- 2006-01
- ISSN（掲載誌）
- 08986568
- 出版事項（掲載誌）
- Elsevier BV
- 対象利用者
- 一般
- DOI
- 10.1016/j.cellsig.2005.03.023
- 作成日（W3CDTF）
- 2005-06-01
- 著作権情報
- https://www.elsevier.com/tdm/userlicense/1.0/
- 関連情報（URI）
- 参照
- Hearing vulnerability after noise exposure in a mouse model of reactive oxygen species overproductionCharacteristics and roles of the volume-sensitive outwardly rectifying (VSOR) anion channel in the central nervous systemTyrosine kinase FYN negatively regulates NOX4 in cardiac remodelingPharmacological inhibition of TLR4-NOX4 signal protects against neuronal death in transient focal ischemiaConstitutive activity of NADPH oxidase 1 (Nox1) that promotes its own activity suppresses the colon epithelial cell migrationAzithromycin attenuates myofibroblast differentiation and lung fibrosis development through proteasomal degradation of NOX4The downregulation of NADPH oxidase Nox4 during hypoxia in hemangioendothelioma cells: a possible role of p22<i><sup>phox</sup></i> on Nox4 protein stabilityHypoxia stabilizes the <scp>H<sub>2</sub>O<sub>2</sub></scp>‐producing oxidase Nox4 in cardiomyocytes via suppressing autophagy‐related lysosomal degradationStructure, regulation and evolution of Nox‐family NADPH oxidases that produce reactive oxygen speciesA mitochondrial <scp>ROS</scp> pathway controls matrix metalloproteinase 9 levels and invasive properties in <scp>RAS</scp>‐activated cancer cellsIntrahepatic microcirculatory disorder, parenchymal hypoxia and NOX4 upregulation result in zonal differences in hepatocyte apoptosis following lipopolysaccharide- and D-galactosamine-induced acute liver failure in ratsKetone body and FGF21 coordinately regulate fasting-induced oxidative stress response in the heartCardiac fibroblasts regulate the development of heart failure via Htra3-TGF-β-IGFBP7 axisUpregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac MyocytesIncreased Oxidative Stress in the Nucleus Caused by Nox4 Mediates Oxidation of HDAC4 and Cardiac HypertrophyMetformin attenuates lung fibrosis development via NOX4 suppressionRac1-Mediated Activation of Mineralocorticoid Receptor in Pressure Overload–Induced Cardiac InjuryThe NADPH oxidase NOX4 promotes the directed migration of endothelial cells by stabilizing vascular endothelial growth factor receptor 2 proteinSimultaneous angiotensin receptor blockade and glucagon‐like peptide‐1 receptor activation ameliorate albuminuria in obese insulin‐resistant ratsPathophysiological Roles of NADPH Oxidase/Nox Family Proteins in the Vascular System - Review and Perspective -NADPH Oxidase Isoforms and Anti-hypertensive Effects of Atorvastatin Demonstrated in Two Animal Models
- 連携機関・データベース
- 国立情報学研究所 : CiNii Research
- 提供元機関・データベース
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