Metabolic regulation of the CCN family genes by glycolysis in chondrocytes

• 資料種別: 記事
• タイトル: Metabolic regulation of the CCN family genes by glycolysis in chondrocytes
• 著者標目: Sho Akashi; Takashi Nishida; Abdellatif El-Seoudi; Masaharu Takigawa; Seiji Iida; Satoshi Kubota; https://orcid.org/0000-0003-0795-5072
• 出版年月日等: 2017-11-11
• 出版年（W3CDTF）: 2017-11-11
• タイトル（掲載誌）: Journal of Cell Communication and Signaling
• 巻号年月日等（掲載誌）: 12 1
• 掲載巻: 12
• 掲載号: 1
• 掲載ページ: 245-252
• 掲載年月日（W3CDTF）: 2017-11-11
• ISSN（掲載誌）: 18739601
• 出版事項（掲載誌）: Wiley
• 対象利用者: 一般
• 標準番号（その他）: PMID : 29129024
• DOI: 10.1007/s12079-017-0420-8
• 作成日（W3CDTF）: 2017-11-11
• 著作権情報: http://www.springer.com/tdm
• 関連情報（URI）: http://link.springer.com/article/10.1007/s12079-017-0420-8/fulltext.html; http://link.springer.com/content/pdf/10.1007/s12079-017-0420-8.pdf
• 参照: Regulation of cellular communication factor 2 (CCN2) in breast cancer cells via the cell-type dependent interplay between CCN2 and glycolysis; https://cir.nii.ac.jp/crid/1050007980273838208; Do not overwork: cellular communication network factor 3 for life in cartilage; https://cir.nii.ac.jp/crid/1050017655059038336; CCN3 (NOV) Drives Degradative Changes in Aging Articular Cartilage; https://cir.nii.ac.jp/crid/1050286428527982976; Bipartite regulation of cellular communication network factor 2 and fibroblast growth factor 1 genes by fibroblast growth factor 1 through histone deacetylase 1 and fork head box protein A1; https://cir.nii.ac.jp/crid/1050290769979715200; Molecular and Genetic Interactions between CCN2 and CCN3 behind Their Yin-Yang Collaboration; https://cir.nii.ac.jp/crid/1050292561229694208; RFX1-mediated CCN3 induction that may support chondrocyte survival under starved conditions; https://cir.nii.ac.jp/crid/1050854718070385408; Cellular communication network factor 3 in cartilage development and maintenance; https://cir.nii.ac.jp/crid/1360294643765586176; Cellular Fluorescence Imaging for the Evaluation of Bioactivity of CCN Family Proteins; https://cir.nii.ac.jp/crid/1360298757162037504
• 参照: Roles of heterotypic CCN2/CTGF-CCN3/NOV and homotypic CCN2-CCN2 interactions in expression of the differentiated phenotype of chondrocytes; https://cir.nii.ac.jp/crid/1050283687525992192; CCN Family Member 2/Connective Tissue Growth Factor (CCN2/CTGF) Has Anti-Aging Effects That Protect Articular Cartilage from Age-Related Degenerative Changes; https://cir.nii.ac.jp/crid/1050283688042131328; The role of CCN2 in cartilage and bone development; https://cir.nii.ac.jp/crid/1360004231512931840; Cellular and molecular actions of CCN2/CTGF and its role under physiological and pathological conditions; https://cir.nii.ac.jp/crid/1360004233663473920; The CCN family acting throughout the body: recent research developments; https://cir.nii.ac.jp/crid/1360283694605121024; CCN2 as a Novel Molecule Supporting Energy Metabolism of Chondrocytes; https://cir.nii.ac.jp/crid/1360285705138162176; CCN3-mediated promotion of sulfated proteoglycan synthesis in rat chondrocytes from developing joint heads; https://cir.nii.ac.jp/crid/1360285706489655552; The regenerative effects of CCN2 independent modules on chondrocytes in vitro and osteoarthritis models in vivo; https://cir.nii.ac.jp/crid/1360285706994894848; Structural and Functional Properties of CCN Proteins; https://cir.nii.ac.jp/crid/1360292620215463296; Oligomycin-induced Bioenergetic Adaptation in Cancer Cells with Heterogeneous Bioenergetic Organization; https://cir.nii.ac.jp/crid/1360292621607768960; Novel role of CCN3 that maintains the differentiated phenotype of articular cartilage; https://cir.nii.ac.jp/crid/1360567181232339968; Anti-fibrotic effect of CCN3 accompanied by altered gene expression profile of the CCN family; https://cir.nii.ac.jp/crid/1360567181466375936; Catabolic effects of FGF-1 on chondrocytes and its possible role in osteoarthritis; https://cir.nii.ac.jp/crid/1360848656443138048; CCN3 (NOV) Is a Negative Regulator of CCN2 (CTGF) and a Novel Endogenous Inhibitor of the Fibrotic Pathway in an in Vitro Model of Renal Disease; https://cir.nii.ac.jp/crid/1361137045567742080; Taking aim at the extracellular matrix: CCN proteins as emerging therapeutic targets; https://cir.nii.ac.jp/crid/1361418520731726592; Cooperative Regulation of Chondrocyte Differentiation by CCN2 and CCN3 Shown by a Comprehensive Analysis of the CCN Family Proteins in Cartilage; https://cir.nii.ac.jp/crid/1361699994338823936; Involvement of <i>cis</i>‐acting repressive element(s) in the 3′‐untranslated region of human connective tissue growth factor gene; https://cir.nii.ac.jp/crid/1361981469297885312; CTGF/Hcs24, a hypertrophic chondrocyte‐specific gene product, stimulates proliferation and differentiation, but not hypertrophy of cultured articular chondrocytes; https://cir.nii.ac.jp/crid/1362544418848907904; CCN Proteins; https://cir.nii.ac.jp/crid/1363951793824522240; All in the CCN family: essential matricellular signaling modulators emerge from the bunker; https://cir.nii.ac.jp/crid/1363951793884169088; Connective tissue growth factor coordinates chondrogenesis and angiogenesis during skeletal development; https://cir.nii.ac.jp/crid/1364233268588747776; Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis; https://cir.nii.ac.jp/crid/1364233269923820672; Role of Omentin, Vaspin, Cardiotrophin-1, TWEAK and NOV/CCN3 in Obesity and Diabetes Development; https://cir.nii.ac.jp/crid/1364233270089843456; Effects of CTGF/Hcs24, a Product of a Hypertrophic Chondrocyte-Specific Gene, on the Proliferation and Differentiation of Chondrocytes in Culture1; https://cir.nii.ac.jp/crid/1364233270743214848; Regeneration of Defects in Articular Cartilage in Rat Knee Joints by CCN2 (Connective Tissue Growth Factor); https://cir.nii.ac.jp/crid/1364233270764176512
• 連携機関・データベース: 国立情報学研究所 : CiNii Research; https://cir.nii.ac.jp/
• 提供元機関・データベース: Crossref; https://www.crossref.org; 科学研究費助成事業データベース; https://kaken.nii.ac.jp/ja

• 要約等: The CCN family consists of 6 genes in the mammalian genome and produces multifunctional proteins involved in a variety of biological processes. Recent...