Results of Detailed Investigations Into Stevens-Johnson Syndrome With Severe Ocular Complications

• 資料種別: 記事
• タイトル: Results of Detailed Investigations Into Stevens-Johnson Syndrome With Severe Ocular Complications
• 著者標目: Mayumi Ueta
• 出版年月日等: 2018-11-27
• 出版年（W3CDTF）: 2018-11-27
• タイトル（掲載誌）: Investigative Opthalmology & Visual Science
• 巻号年月日等（掲載誌）: 59 14
• 掲載巻: 59
• 掲載号: 14
• 掲載ページ: DES183-
• 掲載年月日（W3CDTF）: 2018-11-27
• ISSN（掲載誌）: 15525783
• 出版事項（掲載誌）: Association for Research in Vision and Ophthalmology (ARVO)
• 件名標目: Anti-Inflammatory Agents, Non-Steroidal; Keratoconjunctivitis; Polymorphism, Single Nucleotide; Immunity, Innate; Toll-Like Receptor 3; Ikaros Transcription Factor; HLA Antigens; Stevens-Johnson Syndrome; Receptors, Prostaglandin E, EP3 Subtype; Animals; Humans; Dry Eye Syndromes; Genetic Predisposition to Disease; Immunity, Mucosal
• 対象利用者: 一般
• 標準番号（その他）: PMID : 30481825
• DOI: 10.1167/iovs.17-23537; https://doi.org/10.1167/iovs.17-23537
• 作成日（W3CDTF）: 2018-11-27
• 著作権情報: http://creativecommons.org/licenses/by-nc-nd/4.0/
• 関連情報（URI）: http://iovs.arvojournals.org/article.aspx?articleid=2717239
• 参照: Regulation of gene expression by miRNA-455-3p, upregulated in the conjunctival epithelium of patients with Stevens–Johnson syndrome in the chronic stage; https://cir.nii.ac.jp/crid/1360009142872865280; Severe ocular complications of SJS/TEN and associations among pre-onset, acute, and chronic factors: a report from the international ophthalmology collaborative group; https://cir.nii.ac.jp/crid/1360017677510117248; HLA genotypes and cold medicine-induced Stevens–Johnson syndrome/toxic epidermal necrolysis with severe ocular complications: a systematic review and meta-analysis; https://cir.nii.ac.jp/crid/1360290617536363648; Human leukocyte antigen B*0702 is protective against ocular Stevens–Johnson syndrome/toxic epidermal necrolysis in the UK population; https://cir.nii.ac.jp/crid/1360290617536423552; Human leucocyte antigen association of patients with Stevens-Johnson syndrome/toxic epidermal necrolysis with severe ocular complications in Han Chinese; https://cir.nii.ac.jp/crid/1360290617536839168; Association of HLA polymorphisms and acetaminophen-related Steven-Johnson syndrome with severe ocular complications in Thai population; https://cir.nii.ac.jp/crid/1360572092513560448; International Union of Basic and Clinical Pharmacology. CIX. Differences and Similarities between Human and Rodent Prostaglandin E2 Receptors (EP1–4) and Prostacyclin Receptor (IP): Specific Roles in Pathophysiologic Conditions; https://cir.nii.ac.jp/crid/1360572092840690432; Susceptibility Genes and HLA for Cold Medicine-Related SJS/TEN with SOC; https://cir.nii.ac.jp/crid/1360861707396498688; Association of HLA class I and II gene polymorphisms with acetaminophen-related Stevens–Johnson syndrome with severe ocular complications in Japanese individuals; https://cir.nii.ac.jp/crid/1361694368207756416
• 参照: IKZF1, a new susceptibility gene for cold medicine–related Stevens-Johnson syndrome/toxic epidermal necrolysis with severe mucosal involvement; https://cir.nii.ac.jp/crid/1360002215980782464; Human Leukocyte Antigen Class I Genes Associated With Stevens-Johnson Syndrome and Severe Ocular Complications Following Use of Cold Medicine in a Brazilian Population; https://cir.nii.ac.jp/crid/1360004229750598784; Predictive Factors Associated With Acute Ocular Involvement in Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis; https://cir.nii.ac.jp/crid/1360004231949674496; Association between prostaglandin E receptor 3 polymorphisms and Stevens-Johnson syndrome identified by means of a genome-wide association study; https://cir.nii.ac.jp/crid/1360004232228310144; The role of toll-like receptor 3 in chronic contact hypersensitivity induced by repeated elicitation; https://cir.nii.ac.jp/crid/1360004232266360448; Prostaglandin E Receptor Subtype EP3 Expression in Human Conjunctival Epithelium and Its Changes in Various Ocular Surface Disorders; https://cir.nii.ac.jp/crid/1360004237492955904; HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol; https://cir.nii.ac.jp/crid/1360011144267173120; Downregulation of Monocyte Chemoattractant Protein 1 Expression by Prostaglandin E₂ in Human Ocular Surface Epithelium; https://cir.nii.ac.jp/crid/1360285704697646592; HLA-A*02:06 and PTGER3 polymorphism exert additive effects in cold medicine-related Stevens–Johnson syndrome with severe ocular complications; https://cir.nii.ac.jp/crid/1360285708381979136; Independent strong association of HLA-A*02:06 and HLA-B*44:03 with cold medicine-related Stevens-Johnson syndrome with severe mucosal involvement; https://cir.nii.ac.jp/crid/1360285708503455744; HLA-A*0206 with TLR3 Polymorphisms Exerts More than Additive Effects in Stevens-Johnson Syndrome with Severe Ocular Surface Complications; https://cir.nii.ac.jp/crid/1360285712483448064; Diagnosis and Treatment of Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis with Ocular Complications; https://cir.nii.ac.jp/crid/1360292621144705920; Genome-wide association study identifies HLA-A*3101 allele as a genetic risk factor for carbamazepine-induced cutaneous adverse drug reactions in Japanese population; https://cir.nii.ac.jp/crid/1360565167514907264; Suppression of polyI:C-inducible gene expression by EP3 in murine conjunctival epithelium; https://cir.nii.ac.jp/crid/1360567182167600896; Toll-Like Receptor 3 Increases Allergic and Irritant Contact Dermatitis; https://cir.nii.ac.jp/crid/1360567183365366144; Trans-ethnic study confirmed independent associations of HLA-A*02:06 and HLA-B*44:03 with cold medicine-related Stevens-Johnson syndrome with severe ocular surface complications; https://cir.nii.ac.jp/crid/1360567183480712192; Analysis of Ocular Manifestation and Genetic Association of Allopurinol-Induced Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis in South Korea; https://cir.nii.ac.jp/crid/1360567184845272704; Mucocutaneous inflammation in the Ikaros Family Zinc Finger 1‐keratin 5–specific transgenic mice; https://cir.nii.ac.jp/crid/1360567185423113472; Specific HLA Types are Associated with Antiepileptic Drug-Induced Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis in Japanese Subjects; https://cir.nii.ac.jp/crid/1360567188579764224; Methicillin-Resistant Staphylococcus aureus and Methicillin-Resistant Staphylococcus epidermidis Infections in the Cornea; https://cir.nii.ac.jp/crid/1360574093972520960; HLA Antigens in Stevens-Johnson Syndrome With Ocular Involvement; https://cir.nii.ac.jp/crid/1360574095576826368; Etiologic Factors of the Stevens-Johnson Syndrome; https://cir.nii.ac.jp/crid/1360574096496734464; Ocular surface inflammation is regulated by innate immunity; https://cir.nii.ac.jp/crid/1360846641058853760; Medication Use and the Risk of Stevens–Johnson Syndrome or Toxic Epidermal Necrolysis; https://cir.nii.ac.jp/crid/1360855571124482944; Toll-like receptor 3 enhances late-phase reaction of experimental allergic conjunctivitis; https://cir.nii.ac.jp/crid/1360855571331916928; Polyclonality of Staphylococcus epidermidis residing on the healthy ocular surface; https://cir.nii.ac.jp/crid/1361137046255291904; Strong Association Between HLA-A*0206 and Stevens-Johnson Syndrome in the Japanese; https://cir.nii.ac.jp/crid/1361418520880898432; HLA-A*3101 and Carbamazepine-Induced Hypersensitivity Reactions in Europeans; https://cir.nii.ac.jp/crid/1361699993509268480; Prostaglandin E receptor subtype EP3 downregulates TSLP expression in human conjunctival epithelium; https://cir.nii.ac.jp/crid/1361699993759881216; Prostaglandin E2 suppresses polyinosine-polycytidylic acid (polyI:C)-stimulated cytokine production via prostaglandin E2 receptor (EP) 2 and 3 in human conjunctival epithelial cells; https://cir.nii.ac.jp/crid/1361981469156333824; Cold medicine-related Stevens–Johnson syndrome/toxic epidermal necrolysis with severe ocular complications—phenotypes and genetic predispositions; https://cir.nii.ac.jp/crid/1362262943983034240; Chronic ocular complications of Stevens-Johnson syndrome associated with causative medications in Korea; https://cir.nii.ac.jp/crid/1362262945498134912; Gene-expression analysis of polyI:C-stimulated primary human conjunctival epithelial cells; https://cir.nii.ac.jp/crid/1362825895013792256; Toll-like receptor 3 gene polymorphisms in Japanese patients with Stevens-Johnson syndrome; https://cir.nii.ac.jp/crid/1363107369934055936; HLA-B Locus in Japanese Patients with Anti-Epileptics and Allopurinol-Related Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis; https://cir.nii.ac.jp/crid/1363388845803544832; New Grading System for the Evaluation of Chronic Ocular Manifestations in Patients with Stevens–Johnson Syndrome; https://cir.nii.ac.jp/crid/1363670319273660800; The Incidence of Erythema Multiforme, Stevens-Johnson Syndrome, and Toxic Epidermal Necrolysis; https://cir.nii.ac.jp/crid/1363670319477606784; Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis: Assessment of Medication Risks with Emphasis on Recently Marketed Drugs. The EuroSCAR-Study; https://cir.nii.ac.jp/crid/1363670320912202240; A European study of HLA-B in Stevens–Johnson syndrome and toxic epidermal necrolysis related to five high-risk drugs; https://cir.nii.ac.jp/crid/1363951793353294336; Analysis of the Acute Ophthalmic Manifestations of the Erythema Multiforme/Stevens-Johnson Syndrome/Toxic Epidermal Necrolysis Disease Spectrum; https://cir.nii.ac.jp/crid/1363951793549048960; HLA phenotypes and bullous cutaneous reactions to drugs; https://cir.nii.ac.jp/crid/1363951793912363648; Ethnic Differences in the Association Between Human Leukocyte Antigen and Stevens-Johnson Syndrome; https://cir.nii.ac.jp/crid/1363951795946361472; A marker for Stevens–Johnson syndrome; https://cir.nii.ac.jp/crid/1364233269814407936; A novel IkappaB protein, IkappaB-zeta, induced by proinflammatory stimuli, negatively regulates nuclear factor-kappaB in the nuclei; https://cir.nii.ac.jp/crid/1390282679610496128; Genome-wide association study using the ethnicity-specific Japonica array : identification of new susceptibility loci for cold medicine-related Stevens–Johnson syndrome with severe ocular complications; https://cir.nii.ac.jp/crid/1523106605140160768; Innate immunity of the ocular surface; https://cir.nii.ac.jp/crid/1523669554669226240; Triggering of TLR3 by polyI : C in human corneal epithelial cells to induce inflammatory cytokines; https://cir.nii.ac.jp/crid/1573105976310878464; Human conjunctival epithelial cells express functional Toll-like receptor 5; https://cir.nii.ac.jp/crid/1573668924826712704
• 連携機関・データベース: 国立情報学研究所 : CiNii Research; https://cir.nii.ac.jp/
• 提供元機関・データベース: Crossref; https://www.crossref.org; 科学研究費助成事業データベース; https://kaken.nii.ac.jp/ja

• 要約等: Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN) are acute inflammatory vesiculobullous reactions of the mucosa of the ocular surfa...