記事
Balancing the activation state of the endothelium via two distinct TGF-beta type I receptors
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CiNii Research
Balancing the activation state of the endothelium via two distinct TGF-beta type I receptors
- 資料種別
- 記事
- 著者
- M.-J. Goumans
- 出版者
- Springer Science and Business Media LLC
- 出版年
- 2002-04-01
- 資料形態
- デジタル
- 掲載誌名
- The EMBO Journal 21 7
- 掲載ページ
- p.1743-1753
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デジタル
- 資料種別
- 記事
- 著者標目
- 出版年月日等
- 2002-04-01
- 出版年（W3CDTF）
- 2002-04-01
- タイトル（掲載誌）
- The EMBO Journal
- 巻号年月日等（掲載誌）
- 21 7
- 掲載巻
- 21
- 掲載号
- 7
- 掲載ページ
- 1743-1753
- 掲載年月日（W3CDTF）
- 2002-04-01
- ISSN（掲載誌）
- http://id.crossref.org/issn/02614189
- 出版事項（掲載誌）
- Springer Science and Business Media LLC
- 対象利用者
- 一般
- DOI
- 10.1093/emboj/21.7.1743
- 作成日（W3CDTF）
- 2002-07-26
- 著作権情報
- http://doi.wiley.com/10.1002/tdm_license_1.1
- 関連情報（URI）
- 参照
- Colorectal cancer cell-derived microvesicles containing microRNA-1246 promote angiogenesis by activating Smad 1/5/8 signaling elicited by PML down-regulation in endothelial cellsCD105 maintains the thermogenic program of beige adipocytes by regulating Smad2 signalingInterference of E2‐2‐mediated effect in endothelial cells by FAM96B through its limited expression of E2‐2Inhibition of endothelial cell activation by bHLH protein E2-2 and its impairment of angiogenesisMultiple Signaling Pathways Coordinate to Induce a Threshold Response in a Chordate EmbryoBinding of Gtf2i-β/δ transcription factors to the ARMS2 gene leads to increased circulating HTRA1 in AMD patients and in vitroSynthesis of Reactive Sulfur Species in Cultured Vascular Endothelial Cells after Exposure to TGF-β1: Induction of Cystathionine γ-Lyase and Cystathionine β-Synthase Expression Mediated by the ALK5-Smad2/3/4 and ALK5-Smad2/3-ATF4 PathwaysA novel ENG mutation causing impaired co-translational processing of endoglin associated with hereditary hemorrhagic telangiectasiaInhibition of development of laser-induced choroidal neovascularization with suppression of infiltration of macrophages in Smad3-null miceVasohibin‐2 is required for epithelial–mesenchymal transition of ovarian cancer cells by modulating transforming growth factor‐β signalingBiglycan Intensifies ALK5–Smad2/3 Signaling by TGF‐β<sub>1</sub> and Downregulates Syndecan‐4 in Cultured Vascular Endothelial CellsActivation of the TGFβ pathway impairs endothelial to haematopoietic transitionTransforming Growth Factor-β Induces Transcription Factors MafK and Bach1 to Suppress Expression of the Heme Oxygenase-1 GeneSmad2/Smad3 in endothelium is indispensable for vascular stability via S1PR1 and N-cadherin expressionsFGF2-induced Ras/Erk MAPK signalling maintains lymphatic endothelial cell identity by up-regulating endothelial cell-specific gene expression and suppressing TGFβ signalling via Smad2Exploring the contribution of ARMS2 and HTRA1 genetic risk factors in age-related macular degenerationInteractions between neural cells and blood vessels in central nervous system developmentTranscription Factor 7-Like 2 (TCF7L2) Regulates Activin Receptor-Like Kinase 1 (ALK1)/Smad1 Pathway for Development of Diabetic NephropathyTAL1/SCL Relieves the E2-2-Mediated Repression of VEGFR2 Promoter ActivityNew Insights into the Developmental Mechanisms of Coronary Vessels and EpicardiumPhosphatidylinositol 3-Kinase Class II α-Isoform PI3K-C2α Is Required for Transforming Growth Factor β-induced Smad Signaling in Endothelial CellsEarly Enhanced Leucine-Rich <i>α</i>-2-Glycoprotein-1 Expression in Glomerular Endothelial Cells of Type 2 Diabetic Nephropathy Model MiceBrightfield Proximity Ligation Assay Reveals Both Canonical and Mixed Transforming Growth Factor-β/Bone Morphogenetic Protein Smad Signaling Complexes in Tissue SectionsTGF-β1 Potentiates the Cytotoxicity of Cadmium by Induction of a Metal Transporter, ZIP8, Mediated by the ALK5-Smad2/3 and ALK5-Smad3-p38 MAPK Signal Pathways in Cultured Vascular Endothelial CellsReceptor-activated transcription factors and beyond: multiple modes of Smad2/3-dependent transmission of TGF-β signalingPoor vessel formation in embryos from knock-in mice expressing ALK5 with L45 loop mutation defective in Smad activationUpregulation of Robo4 expression by SMAD signaling suppresses vascular permeability and mortality in endotoxemia and COVID-19 modelsTGF-.BETA. Inhibits Vascular Sprouting through TGF-.BETA. type I Receptor in the Mouse Embryonic AortaActivation of TGF-β Receptors and Smad Proteins by Atorvastatin is Related to Reduced Atherogenesis in ApoE/LDLR Double Knockout MiceCholesterol Effects on Endoglin and Its Downstream Pathways in ApoE/LDLR Double Knockout MiceTransforming growth factor-beta and Forkhead box O transcription factors as cardiac fibroblast regulatorsTGF-β1 system in Leydig cells (part 2) TGF-β1 and progesterone, through Smad 1/5, are involved in the hyperplasia/hypertrophy of Leydig cells
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- NII論文ID
- 80015216700