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- 資料種別
- 記事
- 著者・編者
- Hiroshi Sasaki
- 著者標目
- タイトル（掲載誌）
- Journal of radiation research : an official journal of the Japan Radiation Research Society and Japanese Society for Therapeutic Radiology and Oncology
- 巻号年月日等（掲載誌）
- 45(4) 2004.12
- 掲載巻
- 45
- 掲載号
- 4
- 掲載ページ
- 497～508
- 掲載年月日（W3CDTF）
- 2004-12
- ISSN（掲載誌）
- 0449-3060
- ISSN-L（掲載誌）
- 0449-3060
- 出版事項（掲載誌）
- Tokyo : JRR Editorial Committee
- 出版地（国名コード）
- JP
- 本文の言語コード
- eng
- NDLC
- 対象利用者
- 一般
- コレクション（個別）
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > 学術機関 > 学協会
- オンライン閲覧公開範囲
- インターネット公開
- 遠隔複写可否（NDL）
- 不可
- 所蔵機関
- 国立国会図書館
- 請求記号
- Z53-P232
- 関連情報（国立国会図書館永続的識別子）
- info:ndljp/pid/10918619
- 連携機関・データベース
- 国立国会図書館 : 国立国会図書館雑誌記事索引
- 書誌ID（NDLBibID）
- 7201058
- 整理区分コード
- 632
- 要約等
- Lethal Sectoring, Genomic Instability, and Delayed Division Delay in HeLa S3 Cells Surviving Alpha- or X-irradiation*Hiroshi SASAKI Dept. Exp. Radiol., Kyushu Univ. Lethal sectoring is the process for survival in which lethal damage remaining in irradiated cells is eliminated as offspring without reproductive integrity. This process occurs through the postirradiation 1st to 4th divisions with the accompanying appearance of a clonogenic progenitor. The features of lethal sectoring and delayed cell death were explored by analyzing the pedigrees of HeLa cells surviving alpha- (0.45 Gy) or X-irradiation (3 Gy) (20% survival dose). Most (ca.70%) of the lethal damage was eliminated from alpha-particle survivors through the 1st to 2nd divisions, but it persisted in X-ray survivors until the 2nd generation. Nonlethal damage remaining in the clonogenic progenitors led to an elevated incidence of delayed cell death in their progeny. The mean incidence was higher for alpha-particle (16.3%) than X-ray survivors (8.3%), indicating the greater potentiality for genomic instability (GI) by alpha-particles. Misrepaired clustered DNA damage by alpha-particles and unrepaired PLD by X-rays seemed to be involved in the induction of GI. Delayed division delay (DDD) was noticed, though occasionally (ca.10% per cell), with the progeny during the postirradiation 1st-3rd generations. Mean DDD was much longer in alpha- (ca.11 h) than X-irradiated cells (ca.4 h). Supposedly DDD was triggered by delayed chromosome breakage (published in JRR 45: 497-508 [2004]).
- DOI
- 10.11513/jrrsabst.2005.0.278.0
- オンライン閲覧公開範囲
- インターネット公開
- 連携機関・データベース
- 科学技術振興機構 : J-STAGE
- 要約等
- Lethal Sectoring, Genomic Instability, and Delayed Division Delay in HeLa S3 Cells Surviving Alpha- or X-irradiation*Hiroshi SASAKI Dept. Exp. Radiol., Kyushu Univ. Lethal sectoring is the process for survival in which lethal damage remaining in irradiated cells is eliminated as offspring without reproductive integrity. This process occurs through the postirradiation 1st to 4th divisions with the accompanying appearance of a clonogenic progenitor. The features of lethal sectoring and delayed cell death were explored by analyzing the pedigrees of HeLa cells surviving alpha- (0.45 Gy) or X-irradiation (3 Gy) (20% survival dose). Most (ca.70%) of the lethal damage was eliminated from alpha-particle survivors through the 1st to 2nd divisions, but it persisted in X-ray survivors until the 2nd generation. Nonlethal damage remaining in the clonogenic progenitors led to an elevated incidence of delayed cell death in their progeny. The mean incidence was higher for alpha-particle (16.3%) than X-ray survivors (8.3%), indicating the greater potentiality for genomic instability (GI) by alpha-particles. Misrepaired clustered DNA damage by alpha-particles and unrepaired PLD by X-rays seemed to be involved in the induction of GI. Delayed division delay (DDD) was noticed, though occasionally (ca.10% per cell), with the progeny during the postirradiation 1st-3rd generations. Mean DDD was much longer in alpha- (ca.11 h) than X-irradiated cells (ca.4 h). Supposedly DDD was triggered by delayed chromosome breakage (published in JRR 45: 497-508 [2004]).
- DOI
- 10.11513/jrrsabst.2005.0.278.010.1269/jrr.45.497
- オンライン閲覧公開範囲
- インターネット公開
- 関連情報（URI）
- 参照
- A Framework for Analysis of Abortive Colony Size Distributions Using a Model of Branching Processes in Irradiated Normal Human FibroblastsExploration of Over Kill Effect' of High-LET Ar- and Fe-ions by Evaluating the Fraction of Non-hit Cell and Interphase Death
- 参照
- Frequencies of Complex Chromosome Exchange Aberrations Induced by<sup>238</sup>Pu α-particles and Detected by Fluorescence<i>in Situ</i>Hybridization Using Single Chromosome-specific ProbesCell Killing and Division Delay in Asynchronous and Synchronized HeLa Cells Irradiated with Alpha Particles or X RaysRadiation-Induced Lethal Sectoring in YeastDNA Damage during Mitosis in Human Cells Delays the Metaphase/Anaphase Transition via the Spindle-Assembly CheckpointACTION OF X-RAYS ON MAMMALIAN CELLSRange and stopping-power tables for heavy ionsBiological Radiation EffectsDeletion-pattern analysis of α-particle and X-ray induced mutations at the HPRT locus of V79 Chinese hamster cellsProliferation Kinetics of Mouse L-P59 Cells Irradiated with Ultraviolet Light: A Time-Lapse Photographic StudyAnalysis of Ar-ion and X-ray-induced chromatin breakage and repair in V79 plateau-phase cells by the premature chromosome condensation techniqueDependence of Induction of Interphase Death of Chinese Hamster Ovary Cells Exposed to Accelerated Heavy Ions on Linear Energy TransferClusters of DNA Damage Induced by Ionizing Radiation: Formation of Short DNA Fragments. II. Experimental DetectionPersistence of X-Ray-Induced Lethal Sectoring in YeastDelayed Cell Cycle Progression in Human Lymphoblastoid Cells after Exposure to High-LET Radiation Correlates with Extremely Localized DNA DamageTrack Structure Considerations in Low Dose and Low Dose Rate Effects of Ionizing RadiationTransmission of chromosomal instability after plutonium α-particle irradiationRadiation-Induced Genomic Instability: Delayed Mutagenic and Cytogenetic Effects of X Rays and Alpha ParticlesSurvival and Yields of Chromosome Aberrations in Hamster and Human Lung Cells Irradiated by Alpha ParticlesChromosome Condensation May Enhance X-Ray-Related Cell Lethality in a Temperature-Sensitive Mutant (tsBN2) of Baby Hamster Kidney Cells (BHK21)Time-Lapse Cinemicrographic Studies of X-Irradiated HeLa S3 CellsRepair of Potentially Lethal Damage in X-Irradiated HeLa CellsCorrelation of Generation Times in Two Successive Generations of X-Irradiated FM3A CellsA Nuclear Thread Bridging the Sister Cells Prior to Radiation-Induced Cell FusionBinucleate Cell Formation Induced by X IrradiationTime-Lapse Cinemicrographic Studies of X-Irradiated HeLa S3 CellsRadiation Response of Mammalian Cells Grown in Culture: I. Repair of X-Ray Damage in Surviving Chinese Hamster CellsDelayed Chromosomal Instability in Human T-lymphocyte Clones Exposed to Ionizing RadiationDelayed Cell Death, Giant Cell Formation and Chromosome Instability Induced by X-irradiation in Human Embryo CellsRelationship between Aberration Yield and Mitotic Delay in Human Lymphocytes Exposed to 200 MeV/u Fe-ions or X-raysSensitive Detection of LOH Events in a Human Cell Line after C-ion Beam ExposureCinemicrographic and Photographic Observations of HeLa Cells Irradiated with X-rays and Fast NeutronsMultistep Nature of X-ray-induced Neoplastic Transformation in Mammalian Cells: Genetic Alterations and Instability.Heritable Non-lethal Damage to Cultured Human Cells Irradiated with Heavy IonsInduction of chromatin damage and distribution of isochromatid breaks in human fibroblast cells exposed to heavy ion
- 連携機関・データベース
- 国立情報学研究所 : CiNii Research
- 提供元機関・データベース
- Japan Link CenterJapan Link Center雑誌記事索引データベース雑誌記事索引データベースCrossrefPubMed国立国会図書館デジタルコレクションCiNii ArticlesCiNii ArticlesCrossrefCrossref
- 書誌ID（NDLBibID）
- 720105810918619
- NII論文ID
- 110004041053130007000823