記事
HMGB1 Promotes Accelerated Fracture Healing in Traumatic Brain Injury through PINK1/Parkin-Mediated Mitochondrial Autophagy
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HMGB1 Promotes Accelerated Fracture Healing in Traumatic Brain Injury through PINK1/Parkin-Mediated Mitochondrial Autophagy
- 資料種別
- 記事
- 著者
- Chen Shiyangほか
- 出版者
- The Pharmaceutical Society of Japan
- 出版年
- 2024-12-21
- 資料形態
- デジタル
- 掲載誌名
- Biol. Pharm. Bull. 47 12
- 掲載ページ
- p.2143-2153
資料詳細
要約等:
- <p>We aimed to investigate the mechanism of high mobility group box 1 (HMGB1) in the accelerated fracture healing process during Traumatic brain injur...
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デジタル
- 資料種別
- 記事
- 出版年月日等
- 2024-12-21
- 出版年(W3CDTF)
- 2024-12-21
- タイトル(掲載誌)
- Biol. Pharm. Bull.
- 巻号年月日等(掲載誌)
- 47 12
- 掲載巻
- 47
- 掲載号
- 12
- 掲載ページ
- 2143-2153
- 掲載年月日(W3CDTF)
- 2024-12-21
- ISSN(掲載誌)
- 09186158
- 出版事項(掲載誌)
- The Pharmaceutical Society of Japan
- 本文の言語コード
- en
- 件名標目
- 対象利用者
- 一般
- 標準番号(その他)
- PMID : 39710383
- DOI
- 10.1248/bpb.b24-00203
- 関連情報(URI)
- 参照
- Production of a standard closed fracture in laboratory animal boneEpidemiology of Global Pediatric Traumatic Brain Injury: Qualitative ReviewThe Role of HMGB1 in the Pathogenesis of Inflammatory and Autoimmune DiseasesHMGB1 promotes the secretion of multiple cytokines and potentiates the osteogenic differentiation of mesenchymal stem cells through the Ras/MAPK signaling pathwayAnalysis of Post-Traumatic Brain Injury Gene Expression Signature Reveals Tubulins, Nfe2l2, Nfkb, Cd44 and S100a4 as Treatment TargetsModels of traumatic brain injury-highlights and drawbacksAnimal Models of Traumatic Brain Injury and Assessment of Injury SeverityThe presence of intermolecular disulfide cross-links in type III collagen.Engineering Scaffolds Integrated with Calcium Sulfate and Oyster Shell for Enhanced Bone Tissue RegenerationMitochondrial quality control beyond PINK1/ParkinHumoral Factors Enhance Fracture-Healing and Callus Formation in Patients with Traumatic Brain InjuryHMGB1 Inhibition to Ameliorate Organ Failure and Increase Survival in TraumaEffects of pigment epithelium-derived factor on traumatic brain injuryM2 macrophages are closely associated with accelerated clavicle fracture healing in patients with traumatic brain injury: a retrospective cohort studyUse of bone morphogenetic proteins in mesenchymal stem cell stimulation of cartilage and bone repairCD169+ macrophages are critical for osteoblast maintenance and promote intramembranous and endochondral ossification during bone repairSignaling pathways involved in the effects of HMGB1 on mesenchymal stem cell migration and osteoblastic differentiationSelective death of newborn neurons in hippocampal dentate gyrus following moderate experimental traumatic brain injuryExosomes derived from bone marrow mesenchymal stem cells inhibit human aortic vascular smooth muscle cells calcification via the miR-15a/15b/16/NFATc3/OCN axisDoes traumatic brain injury result in accelerated fracture healing?Polydatin mediates Parkin-dependent mitophagy and protects against mitochondria-dependent apoptosis in acute respiratory distress syndromeSmall Shifts in Craniotomy Position in the Lateral Fluid Percussion Injury Model Are Associated with Differential Lesion DevelopmentHMGB1‐induced angiogenesis in perforated disc cells of human temporomandibular jointCallus formation and the rate of healing of femoral fractures in patients with head injuriesHMGB1 promotes bone fracture healing through activation of ERK signaling pathway in a rat tibial fracture modelPINK1/Parkin-mediated mitophagy modulates cadmium-induced apoptosis in rat cerebral cortical neuronsPharmacological agents for bone fracture healing: talking points from recent clinical trialsHMGB1 contributes to osteoarthritis of temporomandibular joint by inducing synovial angiogenesisMitophagy in Traumatic Brain Injury: A New Target for Therapeutic InterventionNeuroinflammation Following Traumatic Brain Injury: Take It Seriously or NotVisualizing and Modulating Mitophagy for Therapeutic Studies of NeurodegenerationExperimental Closed Head Injury: Analysis of Neurological Outcome, Blood–Brain Barrier Dysfunction, Intracranial Neutrophil Infiltration, and Neuronal Cell Death in Mice Deficient in Genes for Pro-Inflammatory CytokinesThe Effect and Regulatory Mechanism of High Mobility Group Box-1 Protein on Immune Cells in Inflammatory DiseasesrAAV‐mediated combined gene transfer and overexpression of TGF‐β and SOX9 remodels human osteoarthritic articular cartilageCyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injuryRecent advances in traumatic brain injuryMdivi-1 alleviates blood-brain barrier disruption and cell death in experimental traumatic brain injury by mitigating autophagy dysfunction and mitophagy activationHead trauma in sports – clinical characteristics, epidemiology and biomarkersBone fracture in a rat femoral fracture model is associated with the activation of autophagyAnimal models of head traumaSplenic Responses in Ischemic Stroke: New Insights into Stroke PathologyOxidative Stress: Major Threat in Traumatic Brain InjuryTraumatic Brain InjuryMedicinal plants and bone healingResponses to cortical injury: I. Methodology and local effects of contusions in the ratPINK1 mutations are associated with sporadic early‐onset parkinsonismThe evidence of low-intensity pulsed ultrasound for in vitro, animal and human fracture healingTargeting Inflammation Driven by HMGB1Intermittent pneumatic compression in fracture and soft-tissue injuries healingCyclosporin A and cardioprotection: from investigative tool to therapeutic agentAutophagy fights disease through cellular self-digestionStage-Specific Secretion of HMGB1 in Cartilage Regulates Endochondral OssificationAntagonism of RAGE suppresses peripheral nerve regenerationAutophagy in the Pathogenesis of DiseaseMechanism of parkin activation by PINK1The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell deathRelationship between heterotopic ossification and traumatic brain injuryMitochondria-targeted cyclosporin A delivery system to treat myocardial ischemia reperfusion injury of ratsLocation is the key to function: HMGB1 in sepsis and trauma-induced inflammationHMGB1-Mediated Neuroinflammatory Responses in Brain Injuries: Potential Mechanisms and Therapeutic OpportunitiesCurrent and future clinical applications of bone morphogenetic proteins in orthopaedic trauma surgeryPINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagyHMGB1 Neutralizing Antibody Attenuates Cardiac Injury and Apoptosis Induced by Hemorrhagic Shock/Resuscitation in RatsCell Death in the Cardiac MyocyteFragility Fractures in Older People in Japan Based on the National Health Insurance Claims Database
- 連携機関・データベース
- 国立情報学研究所 : CiNii Research
- 提供元機関・データベース
- Japan Link CenterCrossrefPubMed