The axhs1/dwf4 auxin-hypersensitive mutant of Arabidopsis thaliana defines a link for integration of auxin and brassinosteroid mediated root elongation
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DOI[10.5511/plantbiotechnology.15.0209a]のデータに遷移します
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- 資料種別
- 記事
- 著者・編者
- Taisuke NagaseHisabumi TakaseJiro Sekiya
- 出版年月日等
- 2015
- 出版年（W3CDTF）
- 2015
- タイトル（掲載誌）
- Plant biotechnology
- 巻号年月日等（掲載誌）
- 32(2)
- 掲載巻
- 32(2)
- ISSN（掲載誌）
- 1347-6114
- ISSN-L（掲載誌）
- 1342-4580
- 本文の言語コード
- eng
- DOI
- 10.5511/plantbiotechnology.15.0209a
- 国立国会図書館永続的識別子
- info:ndljp/pid/11000429
- コレクション（共通）
- コレクション（障害者向け資料：レベル1）
- コレクション（個別）
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > 学術機関 > 学協会
- 収集根拠
- インターネット資料収集保存事業（WARP）
- 受理日（W3CDTF）
- 2017-12-08T10:56:38+09:00
- 保存日（W3CDTF）
- 2015-08-15
- 記録形式（IMT）
- application/pdf
- オンライン閲覧公開範囲
- インターネット公開
- 遠隔複写可否（NDL）
- 不可
- 掲載誌（国立国会図書館永続的識別子）
- info:ndljp/pid/11000424
- 連携機関・データベース
- 国立国会図書館 : 国立国会図書館デジタルコレクション
- コレクション（個別）
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > 学術機関 > 学協会
- オンライン閲覧公開範囲
- インターネット公開
- 遠隔複写可否（NDL）
- 不可
- 所蔵機関
- 国立国会図書館
- 請求記号
- Z54-J126
- 関連情報（国立国会図書館永続的識別子）
- info:ndljp/pid/11000429
- 連携機関・データベース
- 国立国会図書館 : 国立国会図書館雑誌記事索引
- 書誌ID（NDLBibID）
- 026560164
- 整理区分コード
- 632
- 要約等
- To understand the molecular mechanism of auxin-mediated root elongation, we carried out screening to isolate auxin-hypersensitive (<i>axhs</i>) mutants. The T-DNA insertional mutant <i>axhs1</i> was selected on the basis of root phenotypes associated with auxin sensitivity. Results from brassinosteroid (BR) feeding experiments, Tail-PCR and genetic analyses indicate that <i>AXHS1</i> encodes DWF4, which catalyzes the rate limiting step of BR biosynthesis. The <i>axhs1</i> mutant shows increased sensitivity to indole-3-acetic acid (IAA), the synthetic auxin 2,4-dichlorophenoxyacetic acid (2,4-D), auxin transport inhibitors such as 2,3,5-triiodobenzoic acid (TIBA) and N-1-naphthylphthalamic acid (NPA), and the antiauxin p-chlorophenoxyisobutyric acid (PCIB) for the root elongation response. Analysis of the expression of the <i>DR5:GUS</i> and <i>HS:AXR3NT-GUS</i> reporter genes in wild type and <i>axhs1</i> genotypes, and characterization of double mutants between <i>axhs1</i> and mutants affected in auxin biosynthesis (<i>wei2-1</i>), auxin transport (<i>aux1-7</i>, <i>eir1-1</i>) and auxin signal transduction (<i>tir1-1</i>, <i>axr1-3</i>, <i>axr2-1</i>) indicate that auxin hypersensitivity in <i>axhs1</i> is mediated by the auxin-signaling pathway and an AUX1, EIR1/PIN2 dependent auxin uptake.
- DOI
- 10.5511/plantbiotechnology.15.0209a
- オンライン閲覧公開範囲
- インターネット公開
- 連携機関・データベース
- 科学技術振興機構 : J-STAGE
- 要約等
- To understand the molecular mechanism of auxin-mediated root elongation, we carried out screening to isolate auxin-hypersensitive (<i>axhs</i>) mutants. The T-DNA insertional mutant <i>axhs1</i> was selected on the basis of root phenotypes associated with auxin sensitivity. Results from brassinosteroid (BR) feeding experiments, Tail-PCR and genetic analyses indicate that <i>AXHS1</i> encodes DWF4, which catalyzes the rate limiting step of BR biosynthesis. The <i>axhs1</i> mutant shows increased sensitivity to indole-3-acetic acid (IAA), the synthetic auxin 2,4-dichlorophenoxyacetic acid (2,4-D), auxin transport inhibitors such as 2,3,5-triiodobenzoic acid (TIBA) and N-1-naphthylphthalamic acid (NPA), and the antiauxin p-chlorophenoxyisobutyric acid (PCIB) for the root elongation response. Analysis of the expression of the <i>DR5:GUS</i> and <i>HS:AXR3NT-GUS</i> reporter genes in wild type and <i>axhs1</i> genotypes, and characterization of double mutants between <i>axhs1</i> and mutants affected in auxin biosynthesis (<i>wei2-1</i>), auxin transport (<i>aux1-7</i>, <i>eir1-1</i>) and auxin signal transduction (<i>tir1-1</i>, <i>axr1-3</i>, <i>axr2-1</i>) indicate that auxin hypersensitivity in <i>axhs1</i> is mediated by the auxin-signaling pathway and an AUX1, EIR1/PIN2 dependent auxin uptake.
- DOI
- 10.5511/plantbiotechnology.15.0209a
- オンライン閲覧公開範囲
- インターネット公開
- 関連情報（URI）
- 参照
- Sulfonylurea-resistant mutants of Arabidopsis thalianaThe Arabidopsis deetiolated2 Mutant Is Blocked Early in Brassinosteroid BiosynthesisEIR1, a root-specific protein involved in auxin transport, is required for gravitropism in<i>Arabidopsis thaliana</i>Aux/IAA proteins repress expression of reporter genes containing natural and highly active synthetic auxin response elements.Brassinolide Induces <i>IAA5, IAA19</i> , and DR5, a Synthetic Auxin Response Element in Arabidopsis, Implying a Cross Talk Point of Brassinosteroid and Auxin SignalingGenome-Wide Insertional Mutagenesis of <i>Arabidopsis thaliana</i>Efficient isolation and mapping of <i>Arabidopsis thaliana</i> T‐DNA insert junctions by thermal asymmetric interlaced PCRInterdependency of Brassinosteroid and Auxin Signaling in ArabidopsisBrassinosteroids Interact with Auxin to Promote Lateral Root Development in ArabidopsisThe main auxin biosynthesis pathway in <i>Arabidopsis</i><i>p</i>-Chlorophenoxyisobutyric Acid Impairs Auxin Response in Arabidopsis RootAuxin, actin and growth of the <i>Arabidopsis thaliana</i> primary rootAXR1 is involved in BR‐mediated elongation and <i>SAUR‐AC1</i> gene expression in ArabidopsisArabidopsis auxin-resistance gene AXR1 encodes a protein related to ubiquitin-activating enzyme E1Chromosaponin I Specifically Interacts with AUX1 Protein in Regulating the Gravitropic Response of Arabidopsis RootsAuxin stimulates <i>DWARF4</i> expression and brassinosteroid biosynthesis in ArabidopsisAuxin: Regulation, Action, and InteractionMutations in<i>Arabidopsis Multidrug Resistance-Like</i>ABC Transporters Separate the Roles of Acropetal and Basipetal Auxin Transport in Lateral Root DevelopmentGravitropism of<i>Arabidopsis thaliana</i>Roots Requires the Polarization of PIN2 toward the Root Tip in Meristematic Cortical Cells<i>AXR2</i> Encodes a Member of the Aux/IAA Protein FamilyTranscription of DWARF4 Plays a Crucial Role in Auxin-Regulated Root Elongation in Addition to Brassinosteroid Homeostasis in Arabidopsis thalianaA Link between Ethylene and Auxin Uncovered by the Characterization of Two Root-Specific Ethylene-Insensitive Mutants in ArabidopsisAn Arabidopsis Brassinosteroid-Dependent Mutant Is Blocked in Cell ElongationInhibition of Brassinosteroid Biosynthesis by Either a <i>dwarf4</i> Mutation or a Brassinosteroid Biosynthesis Inhibitor Rescues Defects in Tropic Responses of Hypocotyls in the Arabidopsis Mutant <i>nonphototropic hypocotyl 4</i>The Evolving Complexity of the Auxin PathwayAUX1 regulates root gravitropism in Arabidopsis by facilitating auxin uptake within root apical tissuesThe TIR1 protein of <i>Arabidopsis</i> functions in auxin response and is related to human SKP2 and yeast Grr1pAtPIN2 defines a locus of Arabidopsis for root gravitropism controlBRI1/BAK1, a Receptor Kinase Pair Mediating Brassinosteroid SignalingComparison of mechanisms controlling uptake and accumulation of 2,4-dichlorophenoxy acetic acid, naphthalene-1-acetic acid, and indole-3-acetic acid in suspension-cultured tobacco cellsAuxin regulates SCFTIR1-dependent degradation of AUX/IAA proteinsStructure-Function Analysis of the Presumptive Arabidopsis Auxin Permease AUX1[W]Comprehensive Comparison of Auxin-Regulated and Brassinosteroid-Regulated Genes in ArabidopsisYokonolide B, a Novel Inhibitor of Auxin Action, Blocks Degradation of AUX/IAA FactorsLocalization of the auxin permease AUX1 suggests two functionally distinct hormone transport pathways operate in the <i>Arabidopsis</i> root apexBrassinosteroids Stimulate Plant Tropisms through Modulation of Polar Auxin Transport in <i>Brassica</i> and <i>Arabidopsis</i>A Putative Leucine-Rich Repeat Receptor Kinase Involved in Brassinosteroid Signal TransductionAUX1 Promotes Lateral Root Formation by Facilitating Indole-3-Acetic Acid Distribution between Sink and Source Tissues in the Arabidopsis SeedlingReduced naphthylphthalamic acid binding in the tir3 mutant of Arabidopsis is associated with a reduction in polar auxin transport and diverse morphological defects.Arabidopsis <i>Aux/IAA</i> genes are involved in brassinosteroid‐mediated growth responses in a manner dependent on organ type
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- 国立情報学研究所 : CiNii Research
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- 書誌ID（NDLBibID）
- 026560164
- NII論文ID
- 130005085997