Altered DNA methylation in kidney disease : useful markers and therapeutic targets

• 資料種別: 記事
• タイトル: Altered DNA methylation in kidney disease : useful markers and therapeutic targets
• 著者・編者: Kaori Hayashi
• 出版事項: 日本 : Springer Nature
• 出版年月日等: 2022-01-13
• 出版年（W3CDTF）: 2022-01-13
• タイトル（掲載誌）: Clinical and experimental nephrology : official publication of the Japanese Society of Nephrology
• 巻号年月日等（掲載誌）: 26(4)
• 掲載巻: 26(4)
• ISSN（掲載誌）: 1437-7799
• ISSN-L（掲載誌）: 1437-7799
• 本文の言語コード: eng
• DOI: 10.1007/s10157-022-02181-5; https://doi.org/10.1007/s10157-022-02181-5
• 国立国会図書館永続的識別子: info:ndljp/pid/12289747; https://dl.ndl.go.jp/pid/12289747
• コレクション（共通）: 障害者向け資料
• コレクション（障害者向け資料：レベル1）: テキストデータ
• コレクション（個別）: 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他; https://dl.ndl.go.jp/collections/B00035
• 収集根拠: オンライン資料収集制度
• 受理日（W3CDTF）: 2022-05-23T15:55:37+09:00
• 保存日（W3CDTF）: 2022-05-16
• 記録形式（IMT）: application/pdf
• オンライン閲覧公開範囲: 国立国会図書館内限定公開
• デジタル化資料送信: 図書館・個人送信対象外
• 遠隔複写可否（NDL）: 可
• 掲載誌（URI）: https://link.springer.com/article/10.1007/s10157-022-02181-5
• 掲載誌（国立国会図書館永続的識別子）: info:ndljp/pid/12289746; https://dl.ndl.go.jp/pid/12289746
• 連携機関・データベース: 国立国会図書館 : 国立国会図書館デジタルコレクション; https://dl.ndl.go.jp

• 要約等: コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
• DOI: 10.1007/s10157-022-02181-5; https://doi.org/10.1007/s10157-022-02181-5
• オンライン閲覧公開範囲: インターネット公開
• 関連情報（URI）: http://id.ndl.go.jp/digimeta/12289747; https://dl.ndl.go.jp/pid/12289747; https://link.springer.com/content/pdf/10.1007/s10157-022-02181-5.pdf; https://link.springer.com/article/10.1007/s10157-022-02181-5/fulltext.html; https://search.jamas.or.jp/link/ui/2023145749
• 参照: Decreased KAT5 Expression Impairs DNA Repair and Induces Altered DNA Methylation in Kidney Podocytes; https://cir.nii.ac.jp/crid/1360004232044958336; 10-Year Follow-up of Intensive Glucose Control in Type 2 Diabetes; https://cir.nii.ac.jp/crid/1360011143502253696; Feasibility of Regression of Hypertension Using Contemporary Antihypertensive Agents; https://cir.nii.ac.jp/crid/1360011145012476672; FAN1 mutations cause karyomegalic interstitial nephritis, linking chronic kidney failure to defective DNA damage repair; https://cir.nii.ac.jp/crid/1360011145141198464; Podocyte injury and its consequences; https://cir.nii.ac.jp/crid/1360283691035597568; Renin-angiotensin blockade resets podocyte epigenome through Kruppel-like Factor 4 and attenuates proteinuria; https://cir.nii.ac.jp/crid/1360283691771860352; KLF4-dependent epigenetic remodeling modulates podocyte phenotypes and attenuates proteinuria; https://cir.nii.ac.jp/crid/1360283693782918528; Association of glomerular DNA damage and DNA methylation with one-year eGFR decline in IgA nephropathy; https://cir.nii.ac.jp/crid/1360286993354581248; Harnessing Expressed Single Nucleotide Variation and Single Cell RNA Sequencing To Define Immune Cell Chimerism in the Rejecting Kidney Transplant; https://cir.nii.ac.jp/crid/1360294645341104640; Sox2 and Klf4 as the Functional Core in Pluripotency Induction without Exogenous Oct4; https://cir.nii.ac.jp/crid/1360294646828226688; Podocyte-specific KLF4 is required to maintain parietal epithelial cell quiescence in the kidney; https://cir.nii.ac.jp/crid/1360294646870254336; Urinary Single-Cell Profiling Captures the Cellular Diversity of the Kidney; https://cir.nii.ac.jp/crid/1360294647009420672; KLF4 in Macrophages Attenuates TNFα-Mediated Kidney Injury and Fibrosis; https://cir.nii.ac.jp/crid/1360294647080976384; Tumorigenesis: the adaptation of mammalian cells to sustained stress environment by epigenetic alterations and succeeding matched mutations; https://cir.nii.ac.jp/crid/1360294647284966272; The central role of DNA damage in the ageing process; https://cir.nii.ac.jp/crid/1360294647519178240; Inactivation of Apoptosis Antagonizing Transcription Factor in tubular epithelial cells induces accumulation of DNA damage and nephronophthisis; https://cir.nii.ac.jp/crid/1360294648096898560; DNA damage and expression of DNA methylation modulators in urine-derived cells of patients with hypertension and diabetes; https://cir.nii.ac.jp/crid/1360568468332166784; Methylation pattern of urinary DNA as a marker of kidney function decline in diabetes; https://cir.nii.ac.jp/crid/1360572092859500800; Stability and flexibility of epigenetic gene regulation in mammalian development; https://cir.nii.ac.jp/crid/1360574092885453056; Parietal Epithelial Cell Activation Marker in Early Recurrence of FSGS in the Transplant; https://cir.nii.ac.jp/crid/1360574093838801280; A KDM6A–KLF10 reinforcing feedback mechanism aggravates diabetic podocyte dysfunction; https://cir.nii.ac.jp/crid/1360574094565423616; Extracellular Vesicles in Renal Diseases; https://cir.nii.ac.jp/crid/1360574096232634752; DNA repair factor KAT5 prevents ischemic acute kidney injury through glomerular filtration regulation; https://cir.nii.ac.jp/crid/1360576118761896064; The promise of single-cell RNA sequencing for kidney disease investigation; https://cir.nii.ac.jp/crid/1360576120210874240; Epigenome-wide association studies identify DNA methylation associated with kidney function; https://cir.nii.ac.jp/crid/1360576121543001728; Epigenetic regulation of RCAN1 expression in kidney disease and its role in podocyte injury; https://cir.nii.ac.jp/crid/1360576121686904064; Renal Arteriolar Injury by Salt Intake Contributes to Salt Memory for the Development of Hypertension; https://cir.nii.ac.jp/crid/1360848661366349056; Endothelial Krüppel-Like Factor 4 Mediates the Protective Effect of Statins against Ischemic AKI; https://cir.nii.ac.jp/crid/1360848663068441600; Integration of Genetic Testing and Pathology for the Diagnosis of Adults with FSGS; https://cir.nii.ac.jp/crid/1360855569049276288; Krüppel-like factor 4 is a negative regulator of STAT3-induced glomerular epithelial cell proliferation; https://cir.nii.ac.jp/crid/1360855570304608384; Melanoma epigenetics: novel mechanisms, markers, and medicines; https://cir.nii.ac.jp/crid/1360857596879383936; “Pulse” Treatment With High-Dose Angiotensin Blocker Reverses Renal Arteriolar Hypertrophy and Regresses Hypertension; https://cir.nii.ac.jp/crid/1361137043678861824; The Mechanism of Double-Strand DNA Break Repair by the Nonhomologous DNA End-Joining Pathway; https://cir.nii.ac.jp/crid/1361137045219371904; The Diabetes Control and Complications Trial/Epidemiology of Diabetes Interventions and Complications Study at 30 Years: Overview; https://cir.nii.ac.jp/crid/1361137045687761408; DNA methyltransferase 1 may be a therapy target for attenuating diabetic nephropathy and podocyte injury; https://cir.nii.ac.jp/crid/1361137045748177792; Recruitment of DNA methyltransferase I to DNA repair sites; https://cir.nii.ac.jp/crid/1361137046404943232; Proximal tubule ATR regulates DNA repair to prevent maladaptive renal injury responses; https://cir.nii.ac.jp/crid/1361412896471766784; Heterochromatin and the DNA damage response: the need to relaxThis paper is one of a selection of papers in a Special Issue entitled 31st Annual International Asilomar Chromatin and Chromosomes Conference, and has undergone the Journal’s usual peer review process.; https://cir.nii.ac.jp/crid/1361699993538872576; DNA methylation mediates development of HbA1c-associated complications in type 1 diabetes; https://cir.nii.ac.jp/crid/1361981470688012928; Glomerular Aging and Focal Global Glomerulosclerosis; https://cir.nii.ac.jp/crid/1362262945560884480; <scp>DNA</scp> damage in normally and prematurely aged mice; https://cir.nii.ac.jp/crid/1362544420045366656; Dnmt1 and Dnmt3a maintain DNA methylation and regulate synaptic function in adult forebrain neurons; https://cir.nii.ac.jp/crid/1362825895270447232; DNA methylation profile associated with rapid decline in kidney function: findings from the CRIC Study; https://cir.nii.ac.jp/crid/1363107368762913920; Regression of glomerulosclerosis in response to transient treatment with angiotensin II blockers is attenuated by blockade of matrix metalloproteinase-2; https://cir.nii.ac.jp/crid/1363107370528575360; Epigenomic profiling reveals an association between persistence of DNA methylation and metabolic memory in the DCCT/EDIC type 1 diabetes cohort; https://cir.nii.ac.jp/crid/1363388844706647936; Mechanism and regulation of human non-homologous DNA end-joining; https://cir.nii.ac.jp/crid/1363388844814033664; Tip60 functions as a potential corepressor of KLF4 in regulation of HDC promoter activity; https://cir.nii.ac.jp/crid/1363388844871332096; Single-cell transcriptomics of the mouse kidney reveals potential cellular targets of kidney disease; https://cir.nii.ac.jp/crid/1363388845683969152; Feasibility of Treating Prehypertension with an Angiotensin-Receptor Blocker; https://cir.nii.ac.jp/crid/1363670318830076416; Death-associated Protein Kinase 1 Correlates with Podocyte Apoptosis and Renal Damage and Can Be Mediated by miR-361; https://cir.nii.ac.jp/crid/1370294643784987031
• 連携機関・データベース: 国立情報学研究所 : CiNii Research; https://cir.nii.ac.jp/
• 提供元機関・データベース: 雑誌記事索引データベース; https://ndlsearch.ndl.go.jp; Crossref; https://www.crossref.org; 科学研究費助成事業データベース; https://kaken.nii.ac.jp/ja
• 書誌ID（NDLBibID）: 12289747; http://id.ndl.go.jp/bib/12289747