記事
High-resolution genome-wide mapping of HIF-binding sites by ChIP-seq
デジタルデータあり(Crossref)
すぐに読む
CiNii Research
High-resolution genome-wide mapping of HIF-binding sites by ChIP-seq
- 資料種別
- 記事
- 著者
- Johannes Schödelほか
- 出版者
- American Society of Hematology
- 出版年
- 2011-06-09
- 資料形態
- デジタル
- 掲載誌名
- Blood 117 23
- 掲載ページ
- p.e207-e217
資料詳細
要約等:
- <jats:title>Abstract</jats:title><jats:p>Hypoxia-inducible factor (HIF) regulates the major transcriptional cascade central to the response of all mam...
全国の図書館の所蔵
国立国会図書館以外の全国の図書館の所蔵状況を表示します。
所蔵のある図書館から取寄せることが可能かなど、資料の利用方法は、ご自身が利用されるお近くの図書館へご相談ください
書誌情報
この資料の詳細や典拠(同じ主題の資料を指すキーワード、著者名)等を確認できます。
デジタル
- 資料種別
- 記事
- 著者標目
- 出版年月日等
- 2011-06-09
- 出版年(W3CDTF)
- 2011-06-09
- タイトル(掲載誌)
- Blood
- 巻号年月日等(掲載誌)
- 117 23
- 掲載巻
- 117
- 掲載号
- 23
- 掲載ページ
- e207-e217
- 掲載年月日(W3CDTF)
- 2011-06-09
- ISSN(掲載誌)
- 00064971
- 出版事項(掲載誌)
- American Society of Hematology
- 対象利用者
- 一般
- DOI
- 10.1182/blood-2010-10-314427
- 作成日(W3CDTF)
- 2011-03-30
- 参照
- Regulatory mechanisms of hypoxia‐inducible factor 1 activity: Two decades of knowledgeFurospinosulin‐1, Marine Spongean Furanosesterterpene, Suppresses the Growth of Hypoxia‐Adapted Cancer Cells by Binding to Transcriptional Regulators p54<sup>nrb</sup> and LEDGF/p75Role of hypoxia in progressive chronic kidney disease and implications for therapyMolecular characterization and gene expression analysis of hypoxia-inducible factor and its inhibitory factors in kuruma shrimp Marsupenaeus japonicusRegulation of hypoxia-inducible gene expression after HIF activationRegulation of hypoxia‐inducible factor functions in the nucleus by sphingosine‐1‐phosphateNovel Therapeutic Strategy With Hypoxia-Inducible Factors via Reversible Epigenetic Regulation Mechanisms in Progressive Tubulointerstitial FibrosisRevolution of nephrology research by deep sequencing: ChIP-seq and RNA-seqDynamic Change of Chromatin Conformation in Response to Hypoxia Enhances the Expression of GLUT3 (SLC2A3) by Cooperative Interaction of Hypoxia-Inducible Factor 1 and KDM3ATumor hypoxia regulates ganglioside GM3 synthase, which contributes to oxidative stress resistance in malignant melanomaZBTB2 is Recruited to a Specific Subset of HIF-1 Target Loci to Facilitate Full Gene Expression Under HypoxiaDevelopment and pathological changes of neurovascular unit regulated by hypoxia response in the retinaEffect of tissue inhibitor of metalloprotease 1 on human pulp cells <i>in vitro</i> and rat pulp tissue <i>in vivo</i>HIF2α in the uterine stroma permits embryo invasion and luminal epithelium detachmentHypoxia-inducible factor 1 alpha is a poor prognostic factor and potential therapeutic target in malignant peripheral nerve sheath tumorInhibition of the H3K9 methyltransferase G9A attenuates oncogenicity and activates the hypoxia signaling pathwayTranscriptional regulatory elements of hif1α in a distal locus of islet1 in Xenopus laevisMetabolomic Analysis of Small Extracellular Vesicles Derived from Pancreatic Cancer Cells Cultured under Normoxia and HypoxiaHypoxia-inducible factor-1β is essential for upregulation of the hypoxia-induced <i>FLT1</i> gene in placental trophoblastsLiver Regeneration after Hepatectomy and Partial Liver Transplantation<scp>INT6</scp>/<scp>eIF3e</scp> represses E‐cadherin expression through <scp>HIF2α</scp> in lung carcinoma <scp>A549</scp> cellsEndothelial HIF-2 mediates protection and recovery from ischemic kidney injuryHIF-dependent and reversible nucleosome disassembly in hypoxia-inducible gene promotersHIF-KDM3A-MMP12 regulatory circuit ensures trophoblast plasticity and placental adaptations to hypoxiaBioinformatics data mining approach indicates the expression of chromatin immunoprecipitation followed by deep sequencing (ChIP‐Seq)‐based hypoxia‐inducible factor‐1α target genes in periplaque lesions of multiple sclerosisPathogenesis of Reinke's Edema of the Vocal FoldThe Roles of Hypoxia Signaling in the Pathogenesis of Cardiovascular Diseases赤血球造血因子製剤と低酸素誘導因子水酸化酵素阻害薬の使い分け
- 連携機関・データベース
- 国立情報学研究所 : CiNii Research
- 提供元機関・データベース
- CrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossrefCrossref