膵β細胞におけるME...

膵β細胞におけるMEK/ERKシグナル伝達はβ細胞量とグルコース刺激インスリン分泌の両面の機能的調節によりグルコースホメオスタシスを維持する

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膵β細胞におけるMEK/ERKシグナル伝達はβ細胞量とグルコース刺激インスリン分泌の両面の機能的調節によりグルコースホメオスタシスを維持する

Call No. (NDL)
Z74-F696
Bibliographic ID of National Diet Library
032065622
Material type
記事
Author
Yoshiko Matsumoto Ikushimaほか
Publisher
Tokyo : Blackwell Publishing
Publication date
2022-03
Material Format
Paper
Journal name
Diabetes : a journal of the American Diabetes Association 15(2):2022.3
Publication Page
p.13-27
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Paper

Material Type
記事
Author/Editor
Yoshiko Matsumoto Ikushima
Motoharu Awazawa
Naoki Kobayashi
Sho Osonoi
Seiichi Takemiya
Hiroshi Kobayashi
Hirotsugu Suwanai
Yuichi Morimoto
Kotaro Soeda
Jun Adachi
Masafumi Muratani
Jean Charron
Hiroki Mizukami
Noriko Takahashi
Kohjiro Ueki
Alternative Title
MEK/ERK Signaling in β-Cells Bifunctionally Regulates β-Cell Mass and Glucose-Stimulated Insulin Secretion Response to Maintain Glucose Homeostasis
Periodical title
Diabetes : a journal of the American Diabetes Association
No. or year of volume/issue
15(2):2022.3
Volume
15
Issue
2
Pages
13-27