NO ラジカルをイニシエーターとする放射線誘発バイスタンダー効果

Material type: 文書・図像類

Author: 松本, 英樹ほか

Publisher: 篠原出版新社

Publication date: 2001-01

Material Format: Digital

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Note (General)：: Nitric oxide and its metabolites such as peroxynitrite and nitric dioxide cause DNA damage and protein modifications.In addition,nitric oxide can in...

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Material Type: 文書・図像類
Title: NO ラジカルをイニシエーターとする放射線誘発バイスタンダー効果
Author/Editor: 松本, 英樹
加納, 永一
Matsumoto, H
Kano, E
Author Heading: 松本, 英樹
加納, 永一
Matsumoto, H
Kano, E
Publication, Distribution, etc.: 篠原出版新社
Publication Date: 2001-01
Publication Date (W3CDTF): 2001-01
Alternative Title: Contribution of Nitric Oxide to Radiation-Induced Bystander Effect
Periodical title: 癌の臨床
No. or year of volume/issue: 47 1
Volume: 47
Issue: 1
Pages: 8-11
Text Language Code: jpn
Subject Heading: Radiation
Bystander effect
Nitric oxide
p53
Radiotherapy
Target Audience: 一般
Note (General): Nitric oxide and its metabolites such as peroxynitrite and nitric dioxide cause DNA damage and protein modifications.In addition,nitric oxide can induce p53 accumulation.As part of a feedback loop,p53 mediates transcriptional transrepression of inducible nitric oxide synthase.Recent studies have suggested the interactive effects of nitric oxide and p53 on cellular sensitivity against cancer therapeutic agents.This review summarizes current knowledge,including our findings,of the roles of nitric oxide in cellular response to cancer therapeutic agents,in particular radiation-induced bystander effect and the invoIvement of the p53 gene status in this regard.
Access Restrictions: 限定公開
Data Provider (Database): 国立情報学研究所 : 学術機関リポジトリデータベース（IRDB）（機関リポジトリ）
https://irdb.nii.ac.jp
Original Data Provider (Database): 福井大学 : 福井大学学術機関リポジトリ