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Bibliographic Record
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- Material Type
- 規格・テクニカルリポート類
- Title
- Author/Editor
- ルノー三原, フランソワ孫, 怡姫
- Author Heading
- Publication Date
- 2018
- Publication Date (W3CDTF)
- 2018
- Periodical title
- 科学研究費補助金研究成果報告書
- Target Audience
- 一般
- Note (General)
- 出版タイプ： VoRtype:text私たちは、分子XがSCI後に病変のリモデリングを誘発することを発見しましたが、移植されたNSPCの生存率と分布は変わらず、それは私たちがテーマを放棄することにつながりました。#2 瘢痕形成反応性星状細胞の遺伝子発現プロファイルの特徴付けは、他のグループによって報告されています。#3：STAT3が反応性星状細胞およびグリア瘢痕形成の動態を調節するメカニズム (JCBに発表) を説明しました。#4：病理学的神経瘢痕の新規マーカーを特徴付けるために２つの異なる実験モデルが使用された。我々の仮説は、これらの遺伝子は治癒期における不完全な治癒と関連しているということです。 #1: We found that molecule X, regulates the expression of several proteases and induces lesion remodeling after SCI, but the survival rate and distribution of transplanted neural stem cells are unchanged, which led us to abandon the theme. #2 The characterization of the gene expression profile of scar-forming reactive astrocytes has been reported by another research group (Hara M. et al., Nature Medicine, 2017). #3: We found that the transcription factor STAT3 regulates the dynamics of reactive astrocytes in vitro and glial scar formation in vivo. This study was published in the Journal of Cell Biology (Renault-Mihara et al., 2017). #4: Two different experimental models were used to characterize novel markers of pathological nerve scars. Our hypothesis is those genes are associated with incomplete healing in the central nervous system. This study is still in progress.研究種目 : 若手研究(B) 研究期間 : 2016～2018 課題番号 : 16K21360 研究分野 : spinal cord injury