新型SPY-Qシステム心筋血流評価による交感神経刺激心筋保護法の研究

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Author: 富田, 重之ほか

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Publication date: 2014-05-23

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Note (General)：: 金沢大学附属病院本研究の目的は交感神経および、Ganglion Plexusを持続刺激することで、安全な心停止を得ることである。電極間距離3㎜のペン型双極刺激デバイス（25-30Hz，矩形波双極パルス，出力8V，パルス幅0.4ms）を使用し、Ganglion Plexusを左右同時刺激することで安定...

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https://kaken.nii.ac.jp/ja/report/KAKENHI-PROJECT-23592031/23592031seika/

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Material Type: 文書・図像類
Title: 新型SPY-Qシステム心筋血流評価による交感神経刺激心筋保護法の研究
Author/Editor: 富田, 重之
Tomita, Shigeyuki
Author Heading: 富田, 重之
Tomita, Shigeyuki
Publication Date: 2014-05-23
Publication Date (W3CDTF): 2014-05-23
Alternative Title: Study of myocardial protection by sympathetic nerve stimulation using a new SPY-Q system myocardial blood flow evaluation
Periodical title: 平成25(2013)年度科学研究費補助金基盤研究(C) 研究成果報告書 = 2013 Fiscal Year Final Research Report
No. or year of volume/issue: 2011 – 2013
Volume: 2011 – 2013
Pages: 4p.-
Text Language Code: jpn
Subject Heading: 交感神経刺激
高周波刺激
心筋酸素代謝
心筋保護法
Ganglion Plexus
Target Audience: 一般
Note (General): 金沢大学附属病院
本研究の目的は交感神経および、Ganglion Plexusを持続刺激することで、安全な心停止を得ることである。電極間距離3㎜のペン型双極刺激デバイス（25-30Hz，矩形波双極パルス，出力8V，パルス幅0.4ms）を使用し、Ganglion Plexusを左右同時刺激することで安定した心停止を得た。刺激開始後、速やかに心停止し（30分）、刺激中止後約10分で刺激前の脈、血圧に復した。心停止中、心筋酸素飽和度、心筋血流は低下することなく、本研究から交感神経刺激心停止法は、心筋酸素代謝を低下させることなく、結果心機能障害も低減できる有用な方法であると考えられた。
The purpose of this study is to obtain safety cardiac arrest by sustained stimulation of bilateral sympathetic nerve and Ganglion Plexus. Using a pen type bipolar stimulation device (25-30 Hz, rectangular bipolar pulse, output 8 V, pulse width 0.4 ms) with distance between electrodes 3 mm, a stable cardiac arrest was obtained during 30 minutes. During stimulation (30 minutes), cardiac arrest was obtained and after stopping stimulation, heart beating was started immediately. Heart rate and systemic blood pressure were recovered to the controlled value in about 10 minutes. During cardiac arrest, myocardial blood flow and myocardial oxygen saturation were not decreased. The new cardiac protection of the sympathetic nerve stimulation resulted in safe and useful methods without decreasing of cardiac dysfunction and myocardial oxygen metabolism.
研究課題/領域番号:23592031, 研究期間(年度):2011 – 2013
DOI: 10.24517/00059360
Source: https://kanazawa-u.repo.nii.ac.jp/?action=repository_action_common_download&item_id=53076&item_no=1&attribute_id=26&file_no=1
https://kanazawa-u.repo.nii.ac.jp/?action=repository_action_common_download&item_id=53076&item_no=1&attribute_id=26&file_no=1
Access Restrictions: 限定公開
Rights (production): CC BY-NC-ND
Related Material: https://kaken.nii.ac.jp/ja/search/?kw=90334771
https://kaken.nii.ac.jp/ja/grant/KAKENHI-PROJECT-23592031/
https://kaken.nii.ac.jp/ja/report/KAKENHI-PROJECT-23592031/23592031seika/
Data Provider (Database): 国立情報学研究所 : 学術機関リポジトリデータベース（IRDB）（機関リポジトリ）
https://irdb.nii.ac.jp
Original Data Provider (Database): 金沢大学 : 金沢大学学術情報リポジトリKURA