完全ヒト型抗アミノペプチダーゼN抗体であるMT95-4は、マウスモデルにおいて腫瘍の進展を抑制する

Material type: 博士論文

Author: 秋田, 慎

Publisher: -

Publication date: -

Material Format: Digital

Capacity, size, etc.: -

Name of awarding university/degree: 広島大学,博士(医学),Doctor of Philosophy in Medical Science

View All

Related materials as well as pre- and post-revision versions

Cancer Sci 106 (2015) .p921–928

Search by Bookstore

Holdings of Libraries in Japan

This page shows libraries in Japan other than the National Diet Library that hold the material.

List of Cooperating Institutions and Databases

Please contact your local library for information on how to use materials or whether it is possible to request materials from the holding libraries.

other

Hiroshima University Institutional Repository
Digital
You can check the holdings of institutions and databases with which Institutional Repositories DataBase(IRDB)(Institutional Repository) is linked at the site of Institutional Repositories DataBase(IRDB)(Institutional Repository).
Hiroshima University Institutional Repository

Search by Bookstore

Bibliographic Record

You can check the details of this material, its authority (keywords that refer to materials on the same subject, author's name, etc.), etc.

Digital

Material Type: 博士論文
Title: 完全ヒト型抗アミノペプチダーゼN抗体であるMT95-4は、マウスモデルにおいて腫瘍の進展を抑制する
Author/Editor: 秋田, 慎
Author Heading: 秋田, 慎
Alternative Title: MT95-4, a fully humanized antibody raised against aminopeptidase N, reduces tumor progression in a mouse model.
Degree grantor/type: 広島大学
Date Granted: 2015-09-03
Dissertation Number: 甲第6741号
Degree Type: 博士(医学)
Doctor of Philosophy in Medical Science
Text Language Code: eng
Subject Heading: Aminopeptidase N
angiogenesis
fully humanized monoclonal antibody
invasion
tumor growth
Target Audience: 一般
Format (IMT): application/pdf
Source: fulltext
http://ir.lib.hiroshima-u.ac.jp/44053/files/20788
Access Restrictions: インターネット公開
Rights (production): © 2015 The Authors. Cancer Science published by Wiley Publishing Asia Pty Ltd on behalf of Japanese Cancer Association. This is an open access article under the terms of the Creative Commons Attribution-NonCommercial License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.
Related Material: Cancer Sci 106 (2015) .p921–928
Related Material (DOI): 10.1111/cas.12692
Data Provider (Database): 国立情報学研究所 : 学術機関リポジトリデータベース（IRDB）（機関リポジトリ）
https://irdb.nii.ac.jp
Original Data Provider (Database): 広島大学 : 広島大学学術情報リポジトリ