Help
Help
博士論文

A novel mechanism for imatinib mesylate (STI571) resistance in CML cell line KT-1 : role of TC-PTP in modulating signals downstream from the BCR-ABL fusion protein

Icons representing 博士論文

A novel mechanism for imatinib mesylate (STI571) resistance in CML cell line KT-1 : role of TC-PTP in modulating signals downstream from the BCR-ABL fusion protein

Call No. (NDL)
UT51-2005-E126
Bibliographic ID of National Diet Library
000007841907
Material type
博士論文
Author
清水孝恒 [著]
Publisher
[清水孝恒]
Publication date
[2005]
Material Format
Paper
Capacity, size, etc.
2冊
Name of awarding university/degree
慶應義塾大学,博士 (医学)
View All

Notes on use

Note (General)：

博士論文

Search by Bookstore

Search by Bookstore

Search by Bookstore

Bibliographic Record

You can check the details of this material, its authority (keywords that refer to materials on the same subject, author's name, etc.), etc.

Paper

Material Type
博士論文
Title
A novel mechanism for imatinib mesylate (STI571) resistance in CML cell line KT-1 : role of TC-PTP in modulating signals downstream from the BCR-ABL fusion protein
Author/Editor
清水孝恒 [著]
Author Heading
清水, 孝恒 シミズ, タカツネ
Publication, Distribution, etc.
[清水孝恒]
Publication Date
[2005]
Publication Date (W3CDTF)
2005
Extent
2冊
Alternative Title
慢性骨髄性白血病細胞株KT-1における新たなイマチニブ(STI571)耐性化の機構 : BCR-ABL融合蛋白下流のシグナル伝達を修飾するTC-PTPの役割 マンセイ コツズイセイ ハッケツビョウ サイボウカブ KT-1 ニ オケル アラタナ イマチニブ STI571 タイセイカ ノ キコウ : BCR-ABL ユウゴウ タンパク カリュウ ノ シグナル デンタツ オ シュウショクスル TC-PTP ノ ヤクワリ
Degree grantor/type
慶應義塾大学