記事
Pain regulation by non-neuronal cells and inflammation
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CiNii Research
Pain regulation by non-neuronal cells and inflammation
- Material type
- 記事
- Author
- Ru-Rong Jiほか
- Publisher
- American Association for the Advancement of Science (AAAS)
- Publication date
- 2016-11-04
- Material Format
- Digital
- Journal name
- Science 354 6312
- Publication Page
- p.572-577
Detailed bibliographic record
Summary, etc.:
- <jats:p>Acute pain is protective and a cardinal feature of inflammation. Chronic pain after arthritis, nerve injury, cancer, and chemotherapy is assoc...
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Digital
- Material Type
- 記事
- Author Heading
- Publication Date
- 2016-11-04
- Publication Date (W3CDTF)
- 2016-11-04
- Periodical title
- Science
- No. or year of volume/issue
- 354 6312
- Volume
- 354
- Issue
- 6312
- Pages
- 572-577
- Publication date of volume/issue (W3CDTF)
- 2016-11-04
- ISSN (Periodical Title)
- 00368075
- Publication (Periodical Title)
- American Association for the Advancement of Science (AAAS)
- Target Audience
- 一般
- DOI
- 10.1126/science.aaf8924
- Date Created (W3CDTF)
- 2016-11-03
- Related Material (URI)
- Is Referenced By
- Stimulation of nuclear receptor REV-ERBs suppresses production of pronociceptive molecules in cultured spinal astrocytes and ameliorates mechanical hypersensitivity of inflammatory and neuropathic pain of micePain induces stable, active microcircuits in the somatosensory cortex that provide a therapeutic targetConditioned Medium From the Stem Cells of Human Exfoliated Deciduous Teeth Ameliorates Neuropathic Pain in a Partial Sciatic Nerve Ligation ModelAstrocyte‐mediated synapse remodeling in the pathological brainInhibition of peripheral macrophages by nicotinic acetylcholine receptor agonists suppresses spinal microglial activation and neuropathic pain in mice with peripheral nerve injuryInvolvement of lysophosphatidic acid–induced astrocyte activation underlying the maintenance of partial sciatic nerve injury–induced neuropathic painMicroglial diversity in neuropathic painSexually dimorphic effects of pexidartinib on nerve injury‐induced neuropathic pain in miceMacrophage/microglia-producing transient increase of platelet-activating factor is involved in neuropathic painRole of Macrophage-Mediated Toll-like receptor 4–Interleukin-1R Signaling in Ectopic Tongue Pain Associated With Tooth Pulp InflammationIdentification of molecular targets for toxic action by persulfate, an industrial sulfur compoundA crucial role of HMGB1 in orofacial and widespread pain sensitization following partial infraorbital nerve transectionHeat hypersensitivity is attenuated with altered expression level of spinal astrocytes after sciatic nerve injury in TRPV1 knockout miceInhibition of autotaxin activity ameliorates neuropathic pain derived from lumbar spinal canal stenosisAllodynia by Splenocytes From Mice With Acid-Induced Fibromyalgia-Like Generalized Pain and Its Sexual Dimorphic Regulation by Brain MicrogliaMechanical stimulus-evoked signal transduction between keratinocytes and sensory neurons via extracellular ATPMacrophages transfer mitochondria to sensory neurons to resolve inflammatory painTissue-resident M2 macrophages directly contact primary sensory neurons in the sensory ganglia after nerve injuryReview of Caplan (1991) on cell-based therapeutic technology using Mesenchymal Stem CellsSexually dimorphic effects of pexidartinib on nerve injury-induced neuropathic pain in micePeripheral and Central Mechanisms of Persistent Orofacial PainChemogenetic Regulation of CX3CR1-Expressing Microglia Using Gi-DREADD Exerts Sex-Dependent Anti-Allodynic Effects in Mouse Models of Neuropathic PainChemogenetic Activation of CX3CR1-Expressing Spinal Microglia Using Gq-DREADD Elicits Mechanical Allodynia in Male MiceProfiling of fatty acid metabolism in the dorsal root ganglion after peripheral nerve injuryPotential therapeutic targets for the treatment of opioid abuse and painInhibition of Nuclear Receptor Related Orphan Receptor γ Ameliorates Mechanical Hypersensitivity Through the Suppression of Spinal Microglial ActivationRegulation of neuropathic pain by microglial Orai1 channelsNeuronal and microglial mechanisms for neuropathic pain in the spinal dorsal horn and anterior cingulate cortexPharmacological Regulation of Neuropathic Pain Driven by Inflammatory MacrophagesDifferential mRNA expression of neuroinflammatory modulators in the spinal cord and thalamus of type 2 diabetic monkeysSpinal high‐mobility group box‐1 induces long‐lasting mechanical hypersensitivity through the toll‐like receptor 4 and upregulation of interleukin‐1β in activated astrocytesPerineural high-mobility group box 1 induces mechanical hypersensitivity through activation of spinal microglia: Involvement of glutamate-NMDA receptor dependent mechanism in spinal dorsal hornPathogenic mechanisms of lipid mediator lysophosphatidic acid in chronic painExacerbation of cancer pain after administering immune checkpoint inhibitor in a patient taking opioids: A case reportPlastic changes in nociceptive pathways contributing to persistent orofacial painミクログリアの多様性から読み解く神経障害性疼痛Increased H19 Long Non-coding RNA Expression in Schwann Cells in Peripheral Neuropathic PainPathophysiological mechanisms of persistent orofacial painKetogenic Diet Alleviates Mechanical Allodynia in the Models of Inflammatory and Neuropathic Pain in Male MiceRole of Connexins in Chronic Pain and Their Potential as Therapeutic Targets for Next-Generation Analgesics
- Data Provider (Database)
- 国立情報学研究所 : CiNii Research
- Original Data Provider (Database)
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