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Anisomycin, a JNK and p38 activator, suppresses cell-cell junction formation in 2D cultures of K38 mouse keratinocyte cells and reduces claudin-7 expression, with an increase of paracellular permeability in 3D cultures.

Anisomycin, a JNK and p38 activator, suppresses cell-cell junction formation in 2D cultures of K38 mouse keratinocyte cells and reduces claudin-7 expression, with an increase of paracellular permeability in 3D cultures.
デジタル博士論文障害者向け資料あり
Nikaido, Misaki, Otani, Takahito, Kitagawa, Norio, Ogata, Kayoko, Iida, Hiroshi, Anan, Hisashi, Inai, Tetsuichiro2019-03-02
国立国会図書館
  • 並列タイトル等（連結）Anisomycin, a JNK and p38 activator, suppresses cell-cell junction formation in 2D cultures of K38 mouse keratinocyte cells and reduces claudin-7 expression, with an increase of paracellular permeability in 3D cultures.
  • 並列タイトル等Anisomycin, a JNK and p38 activator, suppresses cell-cell junction formation in 2D cultures of K38 mouse keratinocyte cells and reduces claudin-7 expression, with an increase of paracellular permeability in 3D cultures.

Anisomycin, a JNK and p38 activator, suppresses cell-cell junction formation in 2D cultures of K38 mouse keratinocyte cells and reduces claudin-7 expression, with an increase of paracellular permeability in 3D cultures.

Anisomycin, a JNK and p38 activator, suppresses cell-cell junction formation in 2D cultures of K38 mouse keratinocyte cells and reduces claudin-7 expression, with an increase of paracellular permeability in 3D cultures.
デジタル博士論文
Nikaido, Misaki, Otani, Takahito, Kitagawa, Norio, Ogata, Kayoko, Iida, Hiroshi, Anan, Hisashi, Inai, Tetsuichiro
インターネットで読める全国の図書館

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