Effect of heterozygous deletion of WNK1 on the WNK-OSR1/SPAK-NCC/NKCC1/NKCC2 signal cascade in the kidney and blood vessels

• 資料種別: 記事
• タイトル: Effect of heterozygous deletion of WNK1 on the WNK-OSR1/SPAK-NCC/NKCC1/NKCC2 signal cascade in the kidney and blood vessels
• 著者・編者: Koichiro Susa; Satomi Kita; Takahiro Iwamoto
• 出版事項: 日本 : Springer Nature
• 出版年月日等: 2012-02-01
• 出版年（W3CDTF）: 2012-02-01
• タイトル（掲載誌）: Clinical and experimental nephrology : official publication of the Japanese Society of Nephrology
• 巻号年月日等（掲載誌）: 16(4)
• 掲載巻: 16(4)
• ISSN（掲載誌）: 1437-7799
• ISSN-L（掲載誌）: 1437-7799
• 本文の言語コード: eng
• DOI: 10.1007/s10157-012-0590-x; https://doi.org/10.1007/s10157-012-0590-x
• 国立国会図書館永続的識別子: info:ndljp/pid/11553650; https://dl.ndl.go.jp/pid/11553650
• コレクション（共通）: 障害者向け資料
• コレクション（障害者向け資料：レベル1）: テキストデータ
• コレクション（個別）: 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他; https://dl.ndl.go.jp/collections/B00035
• 収集根拠: オンライン資料収集制度
• 受理日（W3CDTF）: 2020-10-08T21:45:28+09:00
• 保存日（W3CDTF）: 2020-06-18
• 記録形式（IMT）: application/pdf
• オンライン閲覧公開範囲: 国立国会図書館内限定公開
• デジタル化資料送信: 図書館・個人送信対象外
• 遠隔複写可否（NDL）: 可
• 掲載誌（URI）: https://link.springer.com/journal/10157/16/4
• 掲載誌（国立国会図書館永続的識別子）: info:ndljp/pid/11553646; https://dl.ndl.go.jp/pid/11553646
• 連携機関・データベース: 国立国会図書館 : 国立国会図書館デジタルコレクション; https://dl.ndl.go.jp

• 要約等: コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
• DOI: 10.1007/s10157-012-0590-x; https://doi.org/10.1007/s10157-012-0590-x; 10.1016/j.bbrc.2012.09.130; https://doi.org/10.1016/j.bbrc.2012.09.130
• 関連情報（URI）: http://id.ndl.go.jp/digimeta/11553650; https://dl.ndl.go.jp/pid/11553650; https://api.elsevier.com/content/article/PII:S0006291X12019080?httpAccept=text/xml; https://api.elsevier.com/content/article/PII:S0006291X12019080?httpAccept=text/plain; http://link.springer.com/content/pdf/10.1007/s10157-012-0590-x.pdf; http://link.springer.com/article/10.1007/s10157-012-0590-x/fulltext.html; http://link.springer.com/content/pdf/10.1007/s10157-012-0590-x; http://hdl.handle.net/10588/0d4aa60d-68d0-4dfe-b648-c56f34f77673
• 参照: Phosphorylation of Na–Cl cotransporter by OSR1 and SPAK kinases regulates its ubiquitination; https://cir.nii.ac.jp/crid/1360002215823083264; SPAK Deficiency Corrects Pseudohypoaldosteronism II Caused by WNK4 Mutation; https://cir.nii.ac.jp/crid/1360004237528905856; Familial Hyperkalemic Hypertension; https://cir.nii.ac.jp/crid/1360025430643238400; Impaired degradation of WNK by Akt and PKA phosphorylation of KLHL3; https://cir.nii.ac.jp/crid/1360285706978151552; Dietary Salt Intake Regulates WNK3–SPAK–NKCC1 Phosphorylation Cascade in Mouse Aorta Through Angiotensin II; https://cir.nii.ac.jp/crid/1360285711412868352; Discovery of Novel SPAK Inhibitors That Block WNK Kinase Signaling to Cation Chloride Transporters; https://cir.nii.ac.jp/crid/1360285713114919808; Impaired KLHL3-Mediated Ubiquitination of WNK4 Causes Human Hypertension; https://cir.nii.ac.jp/crid/1360565165806503296; Impaired degradation of WNK1 and WNK4 kinases causes PHAII in mutant KLHL3 knock-in mice; https://cir.nii.ac.jp/crid/1360565167515190528; Regulation of blood pressure and renal electrolyte balance by Cullin-RING ligases; https://cir.nii.ac.jp/crid/1360567184854653568; Kelch-Like Protein 2 Mediates Angiotensin II–With No Lysine 3 Signaling in the Regulation of Vascular Tonus; https://cir.nii.ac.jp/crid/1360567188091634432; Regulation of with‐no‐lysine kinase signaling by Kelch‐like proteins; https://cir.nii.ac.jp/crid/1360846643074646400; <i>KLHL3</i> Knockout Mice Reveal the Physiological Role of KLHL3 and the Pathophysiology of Pseudohypoaldosteronism Type II Caused by Mutant KLHL3; https://cir.nii.ac.jp/crid/1360846643399948288; ASK3 responds to osmotic stress and regulates blood pressure by suppressing WNK1-SPAK/OSR1 signaling in the kidney; https://cir.nii.ac.jp/crid/1360848658358826752; WNK1–TAK1 signaling suppresses lipopolysaccharide-induced cytokine production and classical activation in macrophages; https://cir.nii.ac.jp/crid/1360853567802342784
• 参照: Effect of angiotensin II on the WNK-OSR1/SPAK-NCC phosphorylation cascade in cultured mpkDCT cells and in vivo mouse kidney; https://cir.nii.ac.jp/crid/1360002215822810752; Role of the CLOCK Protein in the Mammalian Circadian Mechanism; https://cir.nii.ac.jp/crid/1360011144781244032; Immunolocalization of WNK4 in mouse kidney; https://cir.nii.ac.jp/crid/1360283690288048768; Rhythmic histone acetylation underlies transcription in the mammalian circadian clock; https://cir.nii.ac.jp/crid/1360292619200181760; The WNKs: Atypical Protein Kinases With Pleiotropic Actions; https://cir.nii.ac.jp/crid/1360292620554627200; Diuretics Shift Circadian Rhythm of Blood Pressure From Nondipper to Dipper in Essential Hypertension; https://cir.nii.ac.jp/crid/1360292620561272192; Cloning, genomic organization, alternative splicing and expression analysis of the human gene WNK3 (PRKWNK3); https://cir.nii.ac.jp/crid/1360574094168814464; Genetic Variants of <i>WNK4</i> in Whites and African Americans With Hypertension; https://cir.nii.ac.jp/crid/1360574095211273216; Circadian regulation of renal function; https://cir.nii.ac.jp/crid/1360574095800505344; Circadian rhythm and postural change in natriuresis in non-dipper type of essential hypertension; https://cir.nii.ac.jp/crid/1360574096389035520; Phenotypes of pseudohypoaldosteronism type II caused by the WNK4 D561A missense mutation are dependent on the WNK-OSR1/SPAK kinase cascade; https://cir.nii.ac.jp/crid/1360846644022394496; Acute Insulin Stimulation Induces Phosphorylation of the Na-Cl Cotransporter in Cultured Distal mpkDCT Cells and Mouse Kidney; https://cir.nii.ac.jp/crid/1360846644334688256; Role of the WNK‐activated SPAK kinase in regulating blood pressure; https://cir.nii.ac.jp/crid/1360855568548919424; The WNK1 and WNK4 protein kinases that are mutated in Gordon's hypertension syndrome phosphorylate and activate SPAK and OSR1 protein kinases; https://cir.nii.ac.jp/crid/1360855568952515968; CIRCADIAN VARIATION OF BLOOD-PRESSURE; https://cir.nii.ac.jp/crid/1360855569864206720; Syndrome of hypertension and hyperkalemia with normal glomerular filtration rate.; https://cir.nii.ac.jp/crid/1360855570467838464; Association of Ala589Ser polymorphism of WNK4 gene with essential hypertension in a high-risk Chinese population; https://cir.nii.ac.jp/crid/1360855570506871680; Association of <i>WNK1</i> Gene Polymorphisms and Haplotypes With Ambulatory Blood Pressure in the General Population; https://cir.nii.ac.jp/crid/1360855570919175808; Epithelial Na+ Channel (ENaC), Hormones, and Hypertension; https://cir.nii.ac.jp/crid/1360855570989881984; Endothelial-Specific Expression of WNK1 Kinase Is Essential for Angiogenesis and Heart Development in Mice; https://cir.nii.ac.jp/crid/1361137043755090048; Targeted disruption of the Wnk4 gene decreases phosphorylation of Na-Cl cotransporter, increases Na excretion and lowers blood pressure; https://cir.nii.ac.jp/crid/1361137045592549248; Contractile regulation of the Na⁺-K⁺-2Cl⁻cotransporter in vascular smooth muscle; https://cir.nii.ac.jp/crid/1361418520501787648; Transient Receptor Potential Channels Regulate Myogenic Tone of Resistance Arteries; https://cir.nii.ac.jp/crid/1361699993988505472; The thiazide-sensitive Na-Cl cotransporter is regulated by a WNK kinase signaling complex; https://cir.nii.ac.jp/crid/1361699994243084800; The importance of circadian rhythms on drug response in hypertension and coronary heart disease—from mice and man; https://cir.nii.ac.jp/crid/1361699995713305984; Molecular clock is involved in predictive circadian adjustment of renal function; https://cir.nii.ac.jp/crid/1361699995754685824; From circadian clock gene expression to pathologies; https://cir.nii.ac.jp/crid/1361981468407544064; Diurnal Variations of Blood Pressure and Microalbuminuria in Essential Hypertension; https://cir.nii.ac.jp/crid/1361981470659150976; Pathophysiological roles of WNK kinases in the kidney; https://cir.nii.ac.jp/crid/1362262943631281664; Wnk1 kinase deficiency lowers blood pressure in mice: A gene-trap screen to identify potential targets for therapeutic intervention; https://cir.nii.ac.jp/crid/1362262943933887360; Signaling Mechanisms Underlying the Vascular Myogenic Response; https://cir.nii.ac.jp/crid/1362262945168056064; Functional interactions of the SPAK/OSR1 kinases with their upstream activator WNK1 and downstream substrate NKCC1; https://cir.nii.ac.jp/crid/1362262945903993088; Cardiovascular Expression of the Mouse WNK1 Gene during Development and Adulthood Revealed by a BAC Reporter Assay; https://cir.nii.ac.jp/crid/1362544418421389568; The circadian clock protein Period 1 regulates expression of the renal epithelial sodium channel in mice; https://cir.nii.ac.jp/crid/1362544419582838912; Silent cerebrovascular disease in the elderly. Correlation with ambulatory pressure.; https://cir.nii.ac.jp/crid/1362544419945035648; Distribution and diversity of Na-K-Cl cotransport proteins: a study with monoclonal antibodies; https://cir.nii.ac.jp/crid/1362544420294505984; Human Hypertension Caused by Mutations in WNK Kinases; https://cir.nii.ac.jp/crid/1362544420615427968; Dietary salt regulates the phosphorylation of OSR1/SPAK kinases and the sodium chloride cotransporter through aldosterone; https://cir.nii.ac.jp/crid/1362544421288483968; Expression and phosphorylation of the Na⁺-Cl⁻ cotransporter NCC in vivo is regulated by dietary salt, potassium, and SGK1; https://cir.nii.ac.jp/crid/1362825894771545344; Aldosterone induces circadian gene expression of clock genes in H9c2 cardiomyoblasts; https://cir.nii.ac.jp/crid/1362825894931140992; Regulation of αENaC expression by the circadian clock protein Period 1 in mpkCCDc14 cells; https://cir.nii.ac.jp/crid/1362825895747217664; WNK1 Regulates Vasoconstriction and Blood Pressure Response to α ₁ -Adrenergic Stimulation in Mice; https://cir.nii.ac.jp/crid/1363107368776704640; The Circadian Clock in the Kidney; https://cir.nii.ac.jp/crid/1363107369307136256; Volume sensitivity of cation-Cl ⁻ cotransporters is modulated by the interaction of two kinases: Ste20-related proline-alanine-rich kinase and WNK4; https://cir.nii.ac.jp/crid/1363107369502496384; Myogenic contraction in rat skeletal muscle arterioles: smooth muscle membrane potential and Ca²⁺signaling; https://cir.nii.ac.jp/crid/1363107369710252800; Bimodal regulation of <i>mPeriod</i> promoters by CREB-dependent signaling and CLOCK/BMAL1 activity; https://cir.nii.ac.jp/crid/1363107370942080512; WNK1 Regulates Phosphorylation of Cation-Chloride-coupled Cotransporters via the STE20-related Kinases, SPAK and OSR1; https://cir.nii.ac.jp/crid/1363107370976845696; SPAK-Knockout Mice Manifest Gitelman Syndrome and Impaired Vasoconstriction; https://cir.nii.ac.jp/crid/1363388843319727232; Circadian rhythm of natriuresis is disturbed in nondipper type of essential hypertension; https://cir.nii.ac.jp/crid/1363388843779088896; Short-term stimulation of the thiazide-sensitive Na⁺-Cl⁻cotransporter by vasopressin involves phosphorylation and membrane translocation; https://cir.nii.ac.jp/crid/1363388844024196480; Knockout of Na⁺/Ca²⁺exchanger in smooth muscle attenuates vasoconstriction and L-type Ca²⁺channel current and lowers blood pressure; https://cir.nii.ac.jp/crid/1363388844410694016; Sodium Restriction Shifts Circadian Rhythm of Blood Pressure From Nondipper to Dipper in Essential Hypertension; https://cir.nii.ac.jp/crid/1363670318300575232; Circadian clock genes directly regulate expression of the Na+/H+ exchanger NHE3 in the kidney; https://cir.nii.ac.jp/crid/1363670318642625920; Molecular Pathogenesis of Pseudohypoaldosteronism Type II: Generation and Analysis of a Wnk4 Knockin Mouse Model; https://cir.nii.ac.jp/crid/1363670318845185152; Analysis of Circadian Rhythms in Human Adrenocortical Secretory Activity; https://cir.nii.ac.jp/crid/1363670319168645888; WNK1, a Gene within a Novel Blood Pressure Control Pathway, Tissue-Specifically Generates Radically Different Isoforms with and without a Kinase Domain; https://cir.nii.ac.jp/crid/1363670319220253952; Mouse Models and the Urinary Concentrating Mechanism in the New Millennium; https://cir.nii.ac.jp/crid/1363670319355799296; Aldosterone Stimulates Activity and Surface Expression of NHE3 in Human Primary Proximal Tubule Epithelial Cells (RPTEC); https://cir.nii.ac.jp/crid/1363670319894994944; Coordination of circadian timing in mammals; https://cir.nii.ac.jp/crid/1363670321044968192; Sodium pump α2 subunits control myogenic tone and blood pressure in mice; https://cir.nii.ac.jp/crid/1363951794404228736; The Mammalian Circadian Timing System: Organization and Coordination of Central and Peripheral Clocks; https://cir.nii.ac.jp/crid/1363951794812839680; High Sodium Sensitivity Implicates Nocturnal Hypertension in Essential Hypertension; https://cir.nii.ac.jp/crid/1363951796206332928; Chapter 8 Entrainment pathways and the functional organization of the circadian system; https://cir.nii.ac.jp/crid/1364233269091575040; Circadian changes in plasma renin activity and plasma aldosterone concentration in two-kidney hypertension rats.; https://cir.nii.ac.jp/crid/1364233271079296768; Dippers versus non-dippers; https://cir.nii.ac.jp/crid/1370569247284867076; Localization of bumetanide- and thiazide-sensitive Na–K–Cl cotransporters along the rat nephron; https://cir.nii.ac.jp/crid/1370569247284867088; Circadian expression of the Na+/H+ exchanger NHE3 in the mouse renal medulla; https://cir.nii.ac.jp/crid/1390001204902054656; Regulation of WNK1 kinase by extracellular potassium; https://cir.nii.ac.jp/crid/2051151842050489728
• 連携機関・データベース: 国立情報学研究所 : CiNii Research; https://cir.nii.ac.jp/
• 提供元機関・データベース: 雑誌記事索引データベース; https://ndlsearch.ndl.go.jp; Crossref; https://www.crossref.org; 科学研究費助成事業データベース; https://kaken.nii.ac.jp/ja
• 書誌ID（NDLBibID）: 11553650; http://id.ndl.go.jp/bib/11553650