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Effect of heterozygous deletion of WNK1 on the WNK-OSR1/SPAK-NCC/NKCC1/NKCC2 signal cascade in the kidney and blood vessels

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Effect of heterozygous deletion of WNK1 on the WNK-OSR1/SPAK-NCC/NKCC1/NKCC2 signal cascade in the kidney and blood vessels

Persistent ID (NDL)
info:ndljp/pid/11553650
Material type
記事
Author
Koichiro Susaほか
Publisher
Springer Nature
Publication date
2012-02-01
Material Format
Digital
Journal name
Clinical and experimental nephrology : official publication of the Japanese Society of Nephrology 16(4)
Publication Page
-
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コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他(Provided by: CiNii Research)

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Digital

Material Type
記事
Author/Editor
Koichiro Susa
Satomi Kita
Takahiro Iwamoto
Publication, Distribution, etc.
Publication Date
2012-02-01
Publication Date (W3CDTF)
2012-02-01
Periodical title
Clinical and experimental nephrology : official publication of the Japanese Society of Nephrology
No. or year of volume/issue
16(4)
Volume
16(4)
ISSN (Periodical Title)
1437-7799
ISSN-L (Periodical Title)
1437-7799
Text Language Code
eng
Persistent ID (NDL)
info:ndljp/pid/11553650
Collection (Materials For Handicapped People:1)
Collection (particular)
国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
Acquisition Basis
オンライン資料収集制度
Date Accepted (W3CDTF)
2020-10-08T21:45:28+09:00
Date Captured (W3CDTF)
2020-06-18
Format (IMT)
application/pdf
Access Restrictions
国立国会図書館内限定公開
Service for the Digitized Contents Transmission Service
図書館・個人送信対象外
Availability of remote photoduplication service
Periodical Title (Persistent ID (NDL))
info:ndljp/pid/11553646
Data Provider (Database)
国立国会図書館 : 国立国会図書館デジタルコレクション

Digital

Summary, etc.
コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
Is Referenced By
Phosphorylation of Na–Cl cotransporter by OSR1 and SPAK kinases regulates its ubiquitination
SPAK Deficiency Corrects Pseudohypoaldosteronism II Caused by WNK4 Mutation
Familial Hyperkalemic Hypertension
Impaired degradation of WNK by Akt and PKA phosphorylation of KLHL3
Dietary Salt Intake Regulates WNK3–SPAK–NKCC1 Phosphorylation Cascade in Mouse Aorta Through Angiotensin II
Discovery of Novel SPAK Inhibitors That Block WNK Kinase Signaling to Cation Chloride Transporters
Impaired KLHL3-Mediated Ubiquitination of WNK4 Causes Human Hypertension
Impaired degradation of WNK1 and WNK4 kinases causes PHAII in mutant KLHL3 knock-in mice
Regulation of blood pressure and renal electrolyte balance by Cullin-RING ligases
Kelch-Like Protein 2 Mediates Angiotensin II–With No Lysine 3 Signaling in the Regulation of Vascular Tonus
Regulation of with‐no‐lysine kinase signaling by Kelch‐like proteins
<i>KLHL3</i> Knockout Mice Reveal the Physiological Role of KLHL3 and the Pathophysiology of Pseudohypoaldosteronism Type II Caused by Mutant KLHL3
ASK3 responds to osmotic stress and regulates blood pressure by suppressing WNK1-SPAK/OSR1 signaling in the kidney
WNK1–TAK1 signaling suppresses lipopolysaccharide-induced cytokine production and classical activation in macrophages
References
Effect of angiotensin II on the WNK-OSR1/SPAK-NCC phosphorylation cascade in cultured mpkDCT cells and in vivo mouse kidney
Role of the CLOCK Protein in the Mammalian Circadian Mechanism
Immunolocalization of WNK4 in mouse kidney
Rhythmic histone acetylation underlies transcription in the mammalian circadian clock
The WNKs: Atypical Protein Kinases With Pleiotropic Actions
Diuretics Shift Circadian Rhythm of Blood Pressure From Nondipper to Dipper in Essential Hypertension
Cloning, genomic organization, alternative splicing and expression analysis of the human gene WNK3 (PRKWNK3)
Genetic Variants of <i>WNK4</i> in Whites and African Americans With Hypertension
Circadian regulation of renal function
Circadian rhythm and postural change in natriuresis in non-dipper type of essential hypertension
Phenotypes of pseudohypoaldosteronism type II caused by the WNK4 D561A missense mutation are dependent on the WNK-OSR1/SPAK kinase cascade
Acute Insulin Stimulation Induces Phosphorylation of the Na-Cl Cotransporter in Cultured Distal mpkDCT Cells and Mouse Kidney
Role of the WNK‐activated SPAK kinase in regulating blood pressure
The WNK1 and WNK4 protein kinases that are mutated in Gordon's hypertension syndrome phosphorylate and activate SPAK and OSR1 protein kinases
CIRCADIAN VARIATION OF BLOOD-PRESSURE
Syndrome of hypertension and hyperkalemia with normal glomerular filtration rate.
Association of Ala589Ser polymorphism of WNK4 gene with essential hypertension in a high-risk Chinese population
Association of <i>WNK1</i> Gene Polymorphisms and Haplotypes With Ambulatory Blood Pressure in the General Population
Epithelial Na+ Channel (ENaC), Hormones, and Hypertension
Endothelial-Specific Expression of WNK1 Kinase Is Essential for Angiogenesis and Heart Development in Mice
Targeted disruption of the Wnk4 gene decreases phosphorylation of Na-Cl cotransporter, increases Na excretion and lowers blood pressure
Contractile regulation of the Na<sup>+</sup>-K<sup>+</sup>-2Cl<sup>−</sup>cotransporter in vascular smooth muscle
Transient Receptor Potential Channels Regulate Myogenic Tone of Resistance Arteries
The thiazide-sensitive Na-Cl cotransporter is regulated by a WNK kinase signaling complex
The importance of circadian rhythms on drug response in hypertension and coronary heart disease—from mice and man
Molecular clock is involved in predictive circadian adjustment of renal function
From circadian clock gene expression to pathologies
Diurnal Variations of Blood Pressure and Microalbuminuria in Essential Hypertension
Pathophysiological roles of WNK kinases in the kidney
Wnk1 kinase deficiency lowers blood pressure in mice: A gene-trap screen to identify potential targets for therapeutic intervention
Signaling Mechanisms Underlying the Vascular Myogenic Response
Functional interactions of the SPAK/OSR1 kinases with their upstream activator WNK1 and downstream substrate NKCC1
Cardiovascular Expression of the Mouse WNK1 Gene during Development and Adulthood Revealed by a BAC Reporter Assay
The circadian clock protein Period 1 regulates expression of the renal epithelial sodium channel in mice
Silent cerebrovascular disease in the elderly. Correlation with ambulatory pressure.
Distribution and diversity of Na-K-Cl cotransport proteins: a study with monoclonal antibodies
Human Hypertension Caused by Mutations in WNK Kinases
Dietary salt regulates the phosphorylation of OSR1/SPAK kinases and the sodium chloride cotransporter through aldosterone
Expression and phosphorylation of the Na<sup>+</sup>-Cl<sup>−</sup> cotransporter NCC in vivo is regulated by dietary salt, potassium, and SGK1
Aldosterone induces circadian gene expression of clock genes in H9c2 cardiomyoblasts
Regulation of αENaC expression by the circadian clock protein Period 1 in mpkCCDc14 cells
WNK1 Regulates Vasoconstriction and Blood Pressure Response to α <sub>1</sub> -Adrenergic Stimulation in Mice
The Circadian Clock in the Kidney
Volume sensitivity of cation-Cl <sup>−</sup> cotransporters is modulated by the interaction of two kinases: Ste20-related proline-alanine-rich kinase and WNK4
Myogenic contraction in rat skeletal muscle arterioles: smooth muscle membrane potential and Ca<sup>2+</sup>signaling
Bimodal regulation of <i>mPeriod</i> promoters by CREB-dependent signaling and CLOCK/BMAL1 activity
WNK1 Regulates Phosphorylation of Cation-Chloride-coupled Cotransporters via the STE20-related Kinases, SPAK and OSR1
SPAK-Knockout Mice Manifest Gitelman Syndrome and Impaired Vasoconstriction
Circadian rhythm of natriuresis is disturbed in nondipper type of essential hypertension
Short-term stimulation of the thiazide-sensitive Na<sup>+</sup>-Cl<sup>−</sup>cotransporter by vasopressin involves phosphorylation and membrane translocation
Knockout of Na<sup>+</sup>/Ca<sup>2+</sup>exchanger in smooth muscle attenuates vasoconstriction and L-type Ca<sup>2+</sup>channel current and lowers blood pressure
Sodium Restriction Shifts Circadian Rhythm of Blood Pressure From Nondipper to Dipper in Essential Hypertension
Circadian clock genes directly regulate expression of the Na+/H+ exchanger NHE3 in the kidney
Molecular Pathogenesis of Pseudohypoaldosteronism Type II: Generation and Analysis of a Wnk4 Knockin Mouse Model
Analysis of Circadian Rhythms in Human Adrenocortical Secretory Activity
WNK1, a Gene within a Novel Blood Pressure Control Pathway, Tissue-Specifically Generates Radically Different Isoforms with and without a Kinase Domain
Mouse Models and the Urinary Concentrating Mechanism in the New Millennium
Aldosterone Stimulates Activity and Surface Expression of NHE3 in Human Primary Proximal Tubule Epithelial Cells (RPTEC)
Coordination of circadian timing in mammals
Sodium pump α2 subunits control myogenic tone and blood pressure in mice
The Mammalian Circadian Timing System: Organization and Coordination of Central and Peripheral Clocks
High Sodium Sensitivity Implicates Nocturnal Hypertension in Essential Hypertension
Chapter 8 Entrainment pathways and the functional organization of the circadian system
Circadian changes in plasma renin activity and plasma aldosterone concentration in two-kidney hypertension rats.
Dippers versus non-dippers
Localization of bumetanide- and thiazide-sensitive Na–K–Cl cotransporters along the rat nephron
Circadian expression of the Na+/H+ exchanger NHE3 in the mouse renal medulla
Regulation of WNK1 kinase by extracellular potassium
Data Provider (Database)
国立情報学研究所 : CiNii Research
Original Data Provider (Database)
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Bibliographic ID (NDL)
11553650