Oxidation resistance 1 functions in the maintenance of cellular survival and genome stability in response to oxidative stress-independent DNA damage

• 資料種別: 記事
• タイトル: Oxidation resistance 1 functions in the maintenance of cellular survival and genome stability in response to oxidative stress-independent DNA damage
• 著者・編者: Ako Matsui; Kazunari Hashiguchi; Masao Suzuki
• 出版事項: 日本 : Springer Nature
• 出版年月日等: 2020-11-10
• 出版年（W3CDTF）: 2020-11-10
• タイトル（掲載誌）: Genes and environment
• 巻号年月日等（掲載誌）: 42(29)
• 掲載巻: 42(29)
• ISSN（掲載誌）: 1880-7062
• ISSN-L（掲載誌）: 1880-7046
• 本文の言語コード: eng
• DOI: 10.1186/s41021-020-00168-w; https://doi.org/10.1186/s41021-020-00168-w
• 国立国会図書館永続的識別子: info:ndljp/pid/11616192; https://dl.ndl.go.jp/pid/11616192
• コレクション（共通）: 障害者向け資料
• コレクション（障害者向け資料：レベル1）: テキストデータ
• コレクション（個別）: 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他; https://dl.ndl.go.jp/collections/B00035
• 収集根拠: オンライン資料収集制度
• 受理日（W3CDTF）: 2021-01-13T14:13:05+09:00
• 保存日（W3CDTF）: 2021-01-13
• 記録形式（IMT）: application/pdf
• オンライン閲覧公開範囲: 国立国会図書館内限定公開
• デジタル化資料送信: 図書館・個人送信対象外
• 遠隔複写可否（NDL）: 可
• 掲載誌（URI）: https://genesenvironment.biomedcentral.com/articles
• 掲載誌（国立国会図書館永続的識別子）: info:ndljp/pid/11467640; https://dl.ndl.go.jp/pid/11467640
• 連携機関・データベース: 国立国会図書館 : 国立国会図書館デジタルコレクション; https://dl.ndl.go.jp

• 要約等: コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
• DOI: 10.1186/s41021-020-00168-w; https://doi.org/10.1186/s41021-020-00168-w
• オンライン閲覧公開範囲: 限定公開
• 関連情報（URI）: http://id.ndl.go.jp/digimeta/11616192; https://dl.ndl.go.jp/pid/11616192; http://link.springer.com/content/pdf/10.1186/s41021-020-00168-w.pdf; http://link.springer.com/article/10.1186/s41021-020-00168-w/fulltext.html; https://repo.qst.go.jp/records/81476; https://search.jamas.or.jp/link/ui/2021266970
• 参照: Excess processing of oxidative damaged bases causes hypersensitivity to oxidative stress and low dose rate irradiation.; https://cir.nii.ac.jp/crid/1050564285772215552; Visualisation of γH2AX Foci Caused by Heavy Ion Particle Traversal; Distinction between Core Track versus Non-Track Damage; https://cir.nii.ac.jp/crid/1360004237526700416; DNA damage and repair following treatment with ionizing radiation; https://cir.nii.ac.jp/crid/1360011144497525760; Clustered DNA double-strand break formation and the repair pathway following heavy-ion irradiation; https://cir.nii.ac.jp/crid/1360285709616535808; Balancing repair and tolerance of DNA damage caused by alkylating agents; https://cir.nii.ac.jp/crid/1360292620603475968; Contribution of Indirect Action to Radiation-Induced Mammalian Cell Inactivation: Dependence on Photon Energy and Heavy-Ion LET; https://cir.nii.ac.jp/crid/1360292620816739584; The DNA damage response and cancer therapy; https://cir.nii.ac.jp/crid/1360564064157891840; Role of isolated and clustered DNA damage and the post-irradiating repair process in the effects of heavy ion beam irradiation; https://cir.nii.ac.jp/crid/1360567184592594944; Oxidation resistance 1 is essential for protection against oxidative stress and participates in the regulation of aging in<i>Caenorhabditis elegans</i>; https://cir.nii.ac.jp/crid/1360567189166040448; Biological effect of radiotherapy on cancer cells; https://cir.nii.ac.jp/crid/1360574093963229568; Cell cycle proteins as promising targets in cancer therapy; https://cir.nii.ac.jp/crid/1360574094349369216; Oxidation resistance 1 is a novel senolytic target; https://cir.nii.ac.jp/crid/1360574096548450432; Radiation-induced clustered DNA lesions: Repair and mutagenesis; https://cir.nii.ac.jp/crid/1360846640862860416; Molecular mechanisms of micronucleus, nucleoplasmic bridge and nuclear bud formation in mammalian and human cells; https://cir.nii.ac.jp/crid/1360855569712404096; Functional genomics reveals a family of eukaryotic oxidation protection genes; https://cir.nii.ac.jp/crid/1361137044274847104; The same, only different – DNA damage checkpoints and their reversal throughout the cell cycle; https://cir.nii.ac.jp/crid/1361137044692652928; Oxidative Stress; https://cir.nii.ac.jp/crid/1361137045536229376; DNA breaks and chromosome pulverization from errors in mitosis; https://cir.nii.ac.jp/crid/1361699996141880576; Oxidation resistance 1 prevents genome instability through maintenance of G2/M arrest in gamma-ray-irradiated cells; https://cir.nii.ac.jp/crid/1361975844411710336; Contributions of Direct and Indirect Actions in Cell Killing by High-LET Radiations; https://cir.nii.ac.jp/crid/1361981468981607552; Unrepaired clustered DNA lesions induce chromosome breakage in human cells; https://cir.nii.ac.jp/crid/1361981469899480192; Human OXR1 maintains mitochondrial DNA integrity and counteracts hydrogen peroxide-induced oxidative stress by regulating antioxidant pathways involving p21; https://cir.nii.ac.jp/crid/1362262945788007680; Oxr1 Is Essential for Protection against Oxidative Stress-Induced Neurodegeneration; https://cir.nii.ac.jp/crid/1362825896080907520; Structural/functional analysis of the human OXR1 protein: identification of exon 8 as the anti-oxidant encoding function; https://cir.nii.ac.jp/crid/1363388843826688256; Mutational signatures of redox stress in yeast single-strand DNA and of aging in human mitochondrial DNA share a common feature; https://cir.nii.ac.jp/crid/1363951793523621632; Essential role of <i>BETA2/NeuroD1</i> in development of the vestibular and auditory systems; https://cir.nii.ac.jp/crid/1364233268985901440; TLDc proteins: new players in the oxidative stress response and neurological disease; https://cir.nii.ac.jp/crid/1364233270200883200; Stress Induction and Mitochondrial Localization of Oxr1 Proteins in Yeast and Humans; https://cir.nii.ac.jp/crid/1364233270726857472; The DNA Damage Response: Making It Safe to Play with Knives; https://cir.nii.ac.jp/crid/1364233271024181248; Formation of clustered DNA damage after high-LET irradiation; https://cir.nii.ac.jp/crid/1390282680193854336
• 連携機関・データベース: 国立情報学研究所 : CiNii Research; https://cir.nii.ac.jp/
• 提供元機関・データベース: 雑誌記事索引データベース; https://ndlsearch.ndl.go.jp; Crossref; https://www.crossref.org; 科学研究費助成事業データベース; https://kaken.nii.ac.jp/ja; 学術機関リポジトリデータベース; https://irdb.nii.ac.jp
• 書誌ID（NDLBibID）: 11616192; http://id.ndl.go.jp/bib/11616192