Oxidation resistance 1 functions in the maintenance of cellular survival and genome stability in response to oxidative stress-independent DNA damage
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- Material Type
- 記事
- Author/Editor
- Ako MatsuiKazunari HashiguchiMasao Suzuki
- Publication, Distribution, etc.
- Publication Date
- 2020-11-10
- Publication Date (W3CDTF)
- 2020-11-10
- Periodical title
- Genes and environment
- No. or year of volume/issue
- 42(29)
- Volume
- 42(29)
- ISSN (Periodical Title)
- 1880-7062
- ISSN-L (Periodical Title)
- 1880-7046
- Text Language Code
- eng
- DOI
- 10.1186/s41021-020-00168-w
- Persistent ID (NDL)
- info:ndljp/pid/11616192
- Collection
- Collection (Materials For Handicapped People:1)
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Acquisition Basis
- オンライン資料収集制度
- Date Accepted (W3CDTF)
- 2021-01-13T14:13:05+09:00
- Date Captured (W3CDTF)
- 2021-01-13
- Format (IMT)
- application/pdf
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Periodical Title (URI)
- Periodical Title (Persistent ID (NDL))
- info:ndljp/pid/11467640
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館デジタルコレクション
- Summary, etc.
- コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- DOI
- 10.1186/s41021-020-00168-w
- Access Restrictions
- 限定公開
- Related Material (URI)
- References
- Excess processing of oxidative damaged bases causes hypersensitivity to oxidative stress and low dose rate irradiation.Visualisation of γH2AX Foci Caused by Heavy Ion Particle Traversal; Distinction between Core Track versus Non-Track DamageDNA damage and repair following treatment with ionizing radiationClustered DNA double-strand break formation and the repair pathway following heavy-ion irradiationBalancing repair and tolerance of DNA damage caused by alkylating agentsContribution of Indirect Action to Radiation-Induced Mammalian Cell Inactivation: Dependence on Photon Energy and Heavy-Ion LETThe DNA damage response and cancer therapyRole of isolated and clustered DNA damage and the post-irradiating repair process in the effects of heavy ion beam irradiationOxidation resistance 1 is essential for protection against oxidative stress and participates in the regulation of aging in<i>Caenorhabditis elegans</i>Biological effect of radiotherapy on cancer cellsCell cycle proteins as promising targets in cancer therapyOxidation resistance 1 is a novel senolytic targetRadiation-induced clustered DNA lesions: Repair and mutagenesisMolecular mechanisms of micronucleus, nucleoplasmic bridge and nuclear bud formation in mammalian and human cellsFunctional genomics reveals a family of eukaryotic oxidation protection genesThe same, only different – DNA damage checkpoints and their reversal throughout the cell cycleOxidative StressDNA breaks and chromosome pulverization from errors in mitosisOxidation resistance 1 prevents genome instability through maintenance of G2/M arrest in gamma-ray-irradiated cellsContributions of Direct and Indirect Actions in Cell Killing by High-LET RadiationsUnrepaired clustered DNA lesions induce chromosome breakage in human cellsHuman OXR1 maintains mitochondrial DNA integrity and counteracts hydrogen peroxide-induced oxidative stress by regulating antioxidant pathways involving p21Oxr1 Is Essential for Protection against Oxidative Stress-Induced NeurodegenerationStructural/functional analysis of the human OXR1 protein: identification of exon 8 as the anti-oxidant encoding functionMutational signatures of redox stress in yeast single-strand DNA and of aging in human mitochondrial DNA share a common featureEssential role of <i>BETA2/NeuroD1</i> in development of the vestibular and auditory systemsTLDc proteins: new players in the oxidative stress response and neurological diseaseStress Induction and Mitochondrial Localization of Oxr1 Proteins in Yeast and HumansThe DNA Damage Response: Making It Safe to Play with KnivesFormation of clustered DNA damage after high-LET irradiation
- Data Provider (Database)
- 国立情報学研究所 : CiNii Research
- Original Data Provider (Database)
- 雑誌記事索引データベースCrossref科学研究費助成事業データベース科学研究費助成事業データベース学術機関リポジトリデータベース
- Bibliographic ID (NDL)
- 11616192