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真性赤血球増加症由来JAK2V617F変異体によるシグナル伝達機構の解析

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真性赤血球増加症由来JAK2V617F変異体によるシグナル伝達機構の解析

Material type
文書・図像類
Author
多胡, めぐみ
Publisher
-
Publication date
2010
Material Format
Digital
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type:text真性赤血球増加症の原因遺伝子であるチロシンキナーゼJAK2の点変異体(V617F)が誘導するがん化シグナルの分子機構を解析し、発症機構を分子レベルで理解することを目的とした。JAK2変異体は転写因子STAT5の恒常的な活性化を誘導し、顕著な細胞増殖や腫瘍形成を誘導することを見出した...

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科研費研究者番号 : 30445192

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Digital

Material Type
文書・図像類
Title
真性赤血球増加症由来JAK2V617F変異体によるシグナル伝達機構の解析
Author/Editor
多胡, めぐみ
Author Heading
多胡, めぐみ
Publication Date
2010
Publication Date (W3CDTF)
2010
Alternative Title
シンセイ セッケッキュウ ゾウカショウ ユライ JAK2V617F ヘンイタイ ニ ヨル シグナル デンタツ キコウ ノ カイセキ
Shinsei sekkekkyu zokasho yurai JAK2V617F henitai ni yoru shigunaru dentatsu kiko no kaiseki
Analysis of signal transduction of Polycythemia vera-associated JAK2 V617F mutant
Periodical title
科学研究費補助金研究成果報告書
Text Language Code
jpn
Subject Heading
JAK2,V617F点変異,真性赤血球増加症,STAT5,Akt,Aurora kinase A