Role of Heat Shock Protein 70 in Induction of Stress Fiber Formation in Rat Arterial Endothelial Cells in Response to Stretch Stress

• Material Type: 記事
• Title: Role of Heat Shock Protein 70 in Induction of Stress Fiber Formation in Rat Arterial Endothelial Cells in Response to Stretch Stress
• Title Transcription: Role of Heat Shock Protein 70 in Induction of Stress Fiber Formation in Rat Arterial Endothelial Cells in Response to Stretch Stress
• Author/Editor: Yamashita,Kazuo; Luo,Shan-Shun; Sugimoto,Keiji; Fujii,Sachiko; Takemasa,Tohru; Fu,Song-Bin
• Publication, Distribution, etc.: 日本 : Japan Society of Histochemistry and Cytochemistry
• Publication Date: 2007
• Publication Date (W3CDTF): 2007
• Periodical title: Acta histochemica et cytochemica
• No. or year of volume/issue: 40(1-6)
• Volume: 40(1-6)
• Pages: 9-17
• Text Language Code: ENG
• Subject Heading: heat-shock protein 70; histochemistry; mechanical stretch stress; rat arterial endothelial cells; stress fibers
• Note (General): 著者所属: Department of Molecular Anatomy, Nippon Medical School; 著者所属: Department of Molecular Anatomy, Nippon Medical School:Department of Internal Medicine, Harbin Medical University; 著者所属: Institute of Health and Sports Sciences, University of Tsukuba; 著者所属: Laboratory of Medical Genetics, Harbin Medical University
• Persistent ID (NDL): info:ndljp/pid/10805085; https://dl.ndl.go.jp/pid/10805085
• Collection: 障害者向け資料
• Collection (Materials For Handicapped People:1): テキストデータ
• Collection (particular): 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > 学術機関 > 学協会; https://dl.ndl.go.jp/collections/B00043
• Acquisition Basis: NII-ELS
• Available (W3CDTF): 2017-08-30
• Date Accepted (W3CDTF): 2017-08-22
• Date Captured (W3CDTF): 2017-08-22
• Format (IMT): application/pdf
• Access Restrictions: インターネット公開
• Availability of remote photoduplication service: 不可
• Periodical Title (Persistent ID (NDL)): info:ndljp/pid/10397082; https://dl.ndl.go.jp/pid/10397082
• Data Provider (Database): 国立国会図書館 : 国立国会図書館デジタルコレクション; https://dl.ndl.go.jp

• Summary, etc.: We investigated the mechanism by which endothelial cells (ECs) resist various forms of physical stress using an experimental system consisting of rat ...