Characteristics of coronary artery disease in chronic kidney disease
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DOI[10.1007/s10157-019-01718-5]to the data of the same series
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- Material Type
- 記事
- Author/Editor
- Hideki FujiiKeiji KonoShinichi Nishi
- Publication, Distribution, etc.
- Publication Date
- 2019-03-04
- Publication Date (W3CDTF)
- 2019-03-04
- Periodical title
- Clinical and experimental nephrology : official publication of the Japanese Society of Nephrology
- No. or year of volume/issue
- 23(6)
- Volume
- 23(6)
- ISSN (Periodical Title)
- 1437-7799
- ISSN-L (Periodical Title)
- 1437-7799
- Text Language Code
- eng
- DOI
- 10.1007/s10157-019-01718-5
- Persistent ID (NDL)
- info:ndljp/pid/11657087
- Collection
- Collection (Materials For Handicapped People:1)
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Acquisition Basis
- オンライン資料収集制度
- Date Accepted (W3CDTF)
- 2021-03-31T20:15:18+09:00
- Date Captured (W3CDTF)
- 2021-02-04
- Format (IMT)
- application/pdf
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Periodical Title (URI)
- Periodical Title (Persistent ID (NDL))
- info:ndljp/pid/11657086
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館デジタルコレクション
- Summary, etc.
- Patients with chronic kidney disease (CKD) commonly experience cardiovascular disease (CVD), and a major cause of death in these patients is CVD. Therefore, the prevention of CVD progression is very crucial in patients with CKD. Recently, this relationship between CKD and CVD has increasingly been examined, and a concept termed “cardiorenal syndrome” has been advocated. Coronary artery disease (CAD) and myocardial injury are crucial factors that contribute to the occurrence of CVD. The initial step CAD is endothelial dysfunction that can be detected as a decrease in the coronary flow reserve (CFR). The previous studies have reported that decreased CFR is significantly correlated to coronary events and mortality. Furthermore, CFR reduces with a decline in the kidney function. Another important presentation of CAD is coronary artery calcification. Vascular calcification is a crucial pathophysiological state, particularly in patients with CKD, and it affects the stability of coronary atherosclerotic plaque. In CKD, not only the traditional risk factors but also CKD-related non-traditional risk factors play key roles in CVD progression. Therefore, the mechanisms responsible for CVD progression are very complex; however, their clarification is crucial to improve the prognosis in patients with CKD.
- DOI
- 10.1007/s10157-019-01718-5
- Access Restrictions
- インターネット公開
- Rights (production)
- © The Author(s) 2019.This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
- Related Material (URI)
- Is Referenced By
- Role of Erythropoiesis-Stimulating Agents in Cardiovascular Protection in CKD Patients: Reappraisal of Their Impact and MechanismsCoexistence of Metabolic Dysfunction‐Associated Fatty Liver Disease and Chronic Kidney Disease Is a More Potent Risk Factor for Ischemic Heart DiseaseThe prognostic implications of chronic kidney disease and anemia on long-term outcomes in patients undergoing percutaneous coronary interventionTime to Shift our Mindset to a Phosphate-Centric Approach for Prevention of Cardiovascular Calcification in the Dialysis PopulationImplication of Renal Impairment for Acute Coronary SyndromeDental caries status is associated with arteriosclerosis in patients on hemodialysis
- Data Provider (Database)
- 国立情報学研究所 : CiNii Research
- Original Data Provider (Database)
- 学術機関リポジトリデータベース雑誌記事索引データベースCrossrefCiNii ArticlesCrossrefCrossrefCrossrefCrossrefCrossrefCrossref
- Bibliographic ID (NDL)
- 11657087
- NAID
- 120006634683