Copper-mediated DNA damage caused by purpurin, a natural anthraquinone
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DOI[10.1186/s41021-022-00245-2]to the data of the same series
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- Material Type
- 記事
- Author/Editor
- Hatasu KobayashiYurie MoriRyo Iwasa
- Publication, Distribution, etc.
- Publication Date
- 2022-05-09
- Publication Date (W3CDTF)
- 2022-05-09
- Periodical title
- Genes and environment
- No. or year of volume/issue
- 44(15)
- Volume
- 44(15)
- ISSN (Periodical Title)
- 1880-7062
- ISSN-L (Periodical Title)
- 1880-7046
- Text Language Code
- eng
- DOI
- 10.1186/s41021-022-00245-2
- Persistent ID (NDL)
- info:ndljp/pid/12315106
- Collection
- Collection (Materials For Handicapped People:1)
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Acquisition Basis
- オンライン資料収集制度
- Date Accepted (W3CDTF)
- 2022-08-10T20:17:28+09:00
- Date Captured (W3CDTF)
- 2022-08-08
- Format (IMT)
- application/pdf
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Periodical Title (URI)
- Periodical Title (Persistent ID (NDL))
- info:ndljp/pid/12315091
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館デジタルコレクション
- Summary, etc.
- コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- DOI
- 10.1186/s41021-022-00245-2
- Access Restrictions
- インターネット公開
- Related Material (URI)
- Is Referenced By
- Oxidative DNA Damage by <i>N</i>4-hydroxycytidine, a Metabolite of the SARS-CoV-2 Antiviral MolnupiravirMyricetin causes site-specific DNA damage via reactive oxygen species generation by redox interactions with copper ionsRosmarinic acid, a natural polyphenol, has a potential pro-oxidant risk via NADH-mediated oxidative DNA damage
- References
- Polyphenols with Anti-Amyloid β Aggregation Show Potential Risk of Toxicity Via Pro-Oxidant PropertiesCopper and carcinogenesisPromoting photocatalytic CO2 reduction with a molecular copper purpurin chromophoreMapping of Altromycin B-DNA Adduct at Nucleotide Resolution in the Human Genomic DNA by Ligation-mediated PCRCarcinogenic 3-nitrobenzanthrone induces oxidative damage to isolated and cellular DNAMechanisms of oxidative DNA damage induced by carcinogenic arylaminesToxicity and tumorigenicity of purpurin, a natural hydroxyanthraquinone in rats: induction of bladder neoplasmsThe molecular and cellular basis of copper dysregulation and its relationship with human pathologiesZinc and Copper Levels in Bladder Cancer: A Systematic Review and Meta-AnalysisExcision of DNA adducts of nitrogen mustards by bacterial and mammalian 3-methyladenine-DNA glycosylasesEthylene formation from methionalSpectroscopic and In Silico Evaluation of Interaction of DNA with Six Anthraquinone DerivativesFormation of DNA adducts using nickel(II) complexes of redox-active ligands: a comparison of salen and peptide complexesGenotoxicity of quinone: An insight on DNA adducts and its LC-MS-based detectionCyclic voltammetric studies of 1,2,4‐trihydroxy‐9,10‐anthraquinone, its interaction with calf thymus DNA and anti‐leukemic activity on MOLT‐4 cell lines: a comparison with anthracycline anticancer drugsMass spectrometry investigation of DNA adduct formation from bisphenol A quinone metabolite and MCF-7 cell DNAMechanism of oxidative DNA damage induced by metabolites of carcinogenic naphthaleneMutagenesis by 9,10-anthraquinone derivatives and related compounds in Salmonella typhimuriumThe relationship of copper to DNA damage and damage prevention in humansAnthraquinones: An OverviewPurpurin: A natural anthraquinone with multifaceted pharmacological activitiesCopper and zinc levels in plasma and cancerous tissues and their relation with expression of VEGF and HIF-1 in the pathogenesis of muscle invasive urothelial bladder cancer: a case-controlled clinical studyElectrochemical studies on the binding of a carcinogenic anthraquinone dye, Purpurin (C.I. 58 205) with DNAGenotoxicity of naturally occurring hydroxyanthraquinonesThe role of metals in site-specific DNA damage with reference to carcinogenesis1,2 1Guest Editor: Miral Dizdaroglu 2This article is part of a series of reviews on “Oxidative DNA Damage and Repair.” The full list of papers may be found on the homepage of the journal.Structural basis for recognition and repair of the endogenous mutagen 8-oxoguanine in DNAThe contemporary landscape of occupational bladder cancer within the United Kingdom: a meta‐analysis of risks over the last 80 yearsA new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Insertion of specific bases during DNA synthesis past the oxidation-damaged base 8-oxodG[57] Sequencing end-labeled DNA with base-specific chemical cleavagesFormation of 8-hydroxyguanine moiety in cellular DNA by agents producing oxygen radicals and evidence for its repair8-Hydroxydeoxyguanosine formation at the 5' site of 5'-GG-3' sequences in double-stranded DNA by UV radiation with riboflavin8-Hydroxyguanine, an abundant form of oxidative DNA damage, causes G-T and A-C substitutions.Bathophenanthrolinedisulphonic acid and bathocuproinedisulphonic acid, water soluble reagents for iron and copperComplete nucleotide sequences of the T24 human bladder carcinoma oncogene and its normal homologueContemporary Occupational Carcinogen Exposure and Bladder CancerCritical depurinating DNA adducts: Estrogen adducts in the etiology and prevention of cancer and dopamine adducts in the etiology and prevention of Parkinson's diseaseMechanism of site-specific DNA damage induced by methylhydrazines in the presence of copper(II) or manganese(III)Mechanism of Oxidative DNA Damage Induced by Environmental Carcinogens and Antioxidants8-Hydroxyguanine, a DNA adduct formed by oxygen radicals: its implication on oxygen radical-involved mutagenesis/carcinogenesisSelective enhancing effect of metal ions on mutagenicity
- Data Provider (Database)
- 国立情報学研究所 : CiNii Research
- Original Data Provider (Database)
- 雑誌記事索引データベースCrossref科学研究費助成事業データベース科学研究費助成事業データベース科学研究費助成事業データベース科学研究費助成事業データベースCrossrefCrossrefCrossref
- Bibliographic ID (NDL)
- 12315106