Decreased GM3 correlates with proteinuria in minimal change nephrotic syndrome and focal segmental glomerulosclerosis

• Material Type: 記事
• Title: Decreased GM3 correlates with proteinuria in minimal change nephrotic syndrome and focal segmental glomerulosclerosis
• Author/Editor: Shokichi Naito; Nagako Kawashima; Daisuke Ishii
• Publication, Distribution, etc.: 日本 : Japanese Society of Nephrology
• Publication Date: 2022-07-08
• Publication Date (W3CDTF): 2022-07-08
• Periodical title: Clinical and experimental nephrology : official publication of the Japanese Society of Nephrology
• No. or year of volume/issue: 26(11)
• Volume: 26(11)
• ISSN (Periodical Title): 1437-7799
• ISSN-L (Periodical Title): 1437-7799
• Text Language Code: eng
• DOI: 10.1007/s10157-022-02249-2; https://doi.org/10.1007/s10157-022-02249-2
• Persistent ID (NDL): info:ndljp/pid/12686304; https://dl.ndl.go.jp/pid/12686304
• Collection: 障害者向け資料
• Collection (Materials For Handicapped People:1): テキストデータ
• Collection (particular): 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他; https://dl.ndl.go.jp/collections/B00035
• Acquisition Basis: オンライン資料収集制度
• Date Accepted (W3CDTF): 2023-03-07T17:36:34+09:00
• Date Captured (W3CDTF): 2022-12-20
• Format (IMT): application/pdf
• Access Restrictions: 国立国会図書館内限定公開
• Service for the Digitized Contents Transmission Service: 図書館・個人送信対象外
• Availability of remote photoduplication service: 可
• Periodical Title (URI): https://link.springer.com/article/10.1007/s10157-022-02249-2
• Periodical Title (Persistent ID (NDL)): info:ndljp/pid/12686300; https://dl.ndl.go.jp/pid/12686300
• Data Provider (Database): 国立国会図書館 : 国立国会図書館デジタルコレクション; https://dl.ndl.go.jp

• Summary, etc.: コレクション : 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
• DOI: 10.1007/s10157-022-02249-2; https://doi.org/10.1007/s10157-022-02249-2
• Related Material (URI): http://id.ndl.go.jp/digimeta/12686304; https://dl.ndl.go.jp/pid/12686304; https://link.springer.com/content/pdf/10.1007/s10157-022-02249-2.pdf; https://link.springer.com/article/10.1007/s10157-022-02249-2/fulltext.html; https://search.jamas.or.jp/link/ui/2024000390
• Is Referenced By: Progression of albuminuria and podocyte injury in focal segmental glomerulosclerosis inhibited by enhanced glycosphingolipid GM3 via valproic acid; https://cir.nii.ac.jp/crid/1360021390744198016; Enhanced Levels of Glycosphingolipid GM3 Delay the Progression of Diabetic Nephropathy; https://cir.nii.ac.jp/crid/1360021390749854336; Glycosphingolipid GM3 prevents albuminuria and podocytopathy induced by anti-nephrin antibody; https://cir.nii.ac.jp/crid/1360861707121031424
• References: Depletion of Gangliosides Enhances Articular Cartilage Repair in Mice; https://cir.nii.ac.jp/crid/1050587981431039488; Dissociation of the insulin receptor and caveolin-1 complex by ganglioside GM3 in the state of insulin resistance; https://cir.nii.ac.jp/crid/1360011144058598656; The mystery of membrane organization: composition, regulation and roles of lipid rafts; https://cir.nii.ac.jp/crid/1360011146482671488; Endothelial ganglioside GM3 regulates angiogenesis in solid tumors; https://cir.nii.ac.jp/crid/1360013168763890176; The glomerular parietal epithelial cell’s responses are influenced by SM22 alpha levels; https://cir.nii.ac.jp/crid/1360017285661475712; Gangliosides in Podocyte Biology and Disease; https://cir.nii.ac.jp/crid/1360017287071610752; Renal distribution of ganglioside GM3 in rat models of types 1 and 2 diabetes; https://cir.nii.ac.jp/crid/1360017289326094208; Mice lacking ganglioside GM3 synthase exhibit complete hearing loss due to selective degeneration of the organ of Corti; https://cir.nii.ac.jp/crid/1360292619911864320; Enhanced insulin sensitivity in mice lacking ganglioside GM3; https://cir.nii.ac.jp/crid/1360292621129794944; Roles of adaptor proteins in podocyte biology; https://cir.nii.ac.jp/crid/1360298761429216768; Altered Sphingolipid Metabolism inN-(4-Hydroxyphenyl)- retinamide-resistant A2780 Human Ovarian Carcinoma Cells; https://cir.nii.ac.jp/crid/1360574094350796032; Distribution of glomerular IgG subclass deposits in malignancy-associated membranous nephropathy; https://cir.nii.ac.jp/crid/1360574095359541888; Nephrin is necessary for podocyte recovery following injury in an adult mature glomerulus; https://cir.nii.ac.jp/crid/1360576121859343872; Podocyte histone deacetylase activity regulates murine and human glomerular diseases; https://cir.nii.ac.jp/crid/1360576122109355392; Glycolipid Sialyltransferases Are Enhanced during Neural Differentiation of Mouse Embryonic Carcinoma Cells, P19; https://cir.nii.ac.jp/crid/1360580235269558016; Valproate attenuates the proteinuria, podocyte and renal injury by facilitating autophagy and inactivation of NF-κB/iNOS signaling in diabetic rat; https://cir.nii.ac.jp/crid/1360580235270028416; Characteristics of polyclonal anti-human nephrin antibodies induced by genetic immunization using nephrin cDNA; https://cir.nii.ac.jp/crid/1360580235270870784; Gangliosides as Signaling Regulators in Cancer; https://cir.nii.ac.jp/crid/1360853567501972224; GM3 and cancer; https://cir.nii.ac.jp/crid/1360855570877522048; A New Prospect for the Treatment of Nephrotic Syndrome Based on Network Pharmacology Analysis; https://cir.nii.ac.jp/crid/1360861713865494016; Decreases of ganglioside GM3 in streptozotocin-induced diabetic glomeruli of rats; https://cir.nii.ac.jp/crid/1360861713865858944; ATRA attenuate proteinuria via downregulation of TRPC6 in glomerulosclerosis rats induced by adriamycin; https://cir.nii.ac.jp/crid/1360861713866294912; Gangliosides in Rat Kidney: Composition, Distribution, and Developmental Changes; https://cir.nii.ac.jp/crid/1361137043889356416; Epidermal growth factor receptor tyrosine kinase is modulated by GM3 interaction with N-linked GlcNAc termini of the receptor; https://cir.nii.ac.jp/crid/1361137044695434752; A role for glycosphingolipid accumulation in the renal hypertrophy of streptozotocin-induced diabetes mellitus.; https://cir.nii.ac.jp/crid/1361137045443501568; Ganglioside-mediated modulation of cell growth. Specific effects of GM3 on tyrosine phosphorylation of the epidermal growth factor receptor.; https://cir.nii.ac.jp/crid/1361699994714199040; Glycosphingolipids; https://cir.nii.ac.jp/crid/1361699996118740608; Induction of Glycosphingolipid GM3 Expression by Valproic Acid Suppresses Cancer Cell Growth; https://cir.nii.ac.jp/crid/1361981469274300544; Valproic Acid Attenuates Proteinuria and Kidney Injury; https://cir.nii.ac.jp/crid/1362825896164061440; Nephrin Signaling in the Podocyte: An Updated View of Signal Regulation at the Slit Diaphragm and Beyond; https://cir.nii.ac.jp/crid/1363388843271244928; Tyrosine Kinase Activity of Epidermal Growth Factor Receptor Is Regulated by GM3 Binding through Carbohydrate to Carbohydrate Interactions; https://cir.nii.ac.jp/crid/1363670319861708672; All-<i>trans</i>retinoic acid inhibits the increases in fibronectin and PAI-1 induced by TGF-β1 and Ang II in rat mesangial cells¹; https://cir.nii.ac.jp/crid/1363670319902828032; New insights in glycosphingolipid function: "glycosignaling domain," a cell surface assembly of glycosphingolipids with signal transducer molecules, involved in cell adhesion coupled with signaling; https://cir.nii.ac.jp/crid/1363670320073446784; Podocytopathies; https://cir.nii.ac.jp/crid/1363670321180620160; Soluble FLT1 Binds Lipid Microdomains in Podocytes to Control Cell Morphology and Glomerular Barrier Function; https://cir.nii.ac.jp/crid/1363951796239625216; Diabetic nephropathy induces alterations in the glomerular and tubule lipid profiles; https://cir.nii.ac.jp/crid/1364233268533068160; Glycosynapses: microdomains controlling carbohydrate-dependent cell adhesion and signaling; https://cir.nii.ac.jp/crid/1364233270137773184; Histochemical and immunoelectron microscopic analysis of ganglioside GM3 in human kidney; https://cir.nii.ac.jp/crid/2050307417120962816; New insight into podocyte slit diaphragm, a therapeutic target of proteinuria; https://cir.nii.ac.jp/crid/2050870367078390144; Antigen-binding abilities of anti-nephrin antibody are prescribed by signal sequence of expression vector in genetic immunization; https://cir.nii.ac.jp/crid/2051996266980627328
• Data Provider (Database): 国立情報学研究所 : CiNii Research; https://cir.nii.ac.jp/
• Original Data Provider (Database): 雑誌記事索引データベース; https://ndlsearch.ndl.go.jp; Crossref; https://www.crossref.org; 科学研究費助成事業データベース; https://kaken.nii.ac.jp/ja
• Bibliographic ID (NDL): 12686304; http://id.ndl.go.jp/bib/12686304