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電子書籍・電子雑誌Glycative stress research
Volume number9 (3)
Amyloid be...

Amyloid beta clearance and microglia : effects of glycative stress and melatonin

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Amyloid beta clearance and microglia : effects of glycative stress and melatonin

Call No. (NDL)
Z63-D541
Bibliographic ID of National Diet Library
032834806
Persistent ID (NDL)
info:ndljp/pid/13120781
Material type
記事
Author
Yoshikazu Yoneiほか
Publisher
糖化ストレス研究会
Publication date
2022-09-30
Material Format
Digital
Journal name
Glycative stress research 9(3)
Publication Page
-
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Summary, etc.:

The physiological role of amyloid-β (Aβ) is unknown, while it plays an important role in the onset and progression of Alzheimer's disease (AD). Aβ pol...

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Bibliographic Record

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Digital

Material Type
記事
Author/Editor
Yoshikazu Yonei
Toshio Taira
Shino Otaka
Publication, Distribution, etc.
Publication Date
2022-09-30
Publication Date (W3CDTF)
2022-09-30
Alternative Title
アミロイドβクリアランスとミクログリア : 糖化ストレスおよびメラトニンの影響
Periodical title
Glycative stress research
No. or year of volume/issue
9(3)
Volume
9(3)
ISSN (Periodical Title)
2188-3610
ISSN-L (Periodical Title)
2188-3610
Text Language Code
eng
jpn
Persistent ID (NDL)
info:ndljp/pid/13120781
Collection (Materials For Handicapped People:1)
Collection (particular)
国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
Acquisition Basis
オンライン資料収集制度
Date Accepted (W3CDTF)
2023-12-08T11:19:22+09:00
Date Captured (W3CDTF)
2023-05-20
Format (IMT)
application/pdf
Access Restrictions
国立国会図書館内限定公開
Service for the Digitized Contents Transmission Service
図書館・個人送信対象外
Availability of remote photoduplication service
Periodical Title (Persistent ID (NDL))
info:ndljp/pid/13120776
Data Provider (Database)
国立国会図書館 : 国立国会図書館デジタルコレクション

Digital

Collection (particular)
国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
Access Restrictions
国立国会図書館内限定公開
Service for the Digitized Contents Transmission Service
図書館・個人送信対象外
Availability of remote photoduplication service
Holding library
国立国会図書館
Call No.
Z63-D541
Related Material (Persistent ID (NDL))
info:ndljp/pid/13120781
Data Provider (Database)
国立国会図書館 : 国立国会図書館雑誌記事索引
Bibliographic ID (NDL)
032834806
Bibliographic Record Category (NDL)
632

Digital

Summary, etc.
The physiological role of amyloid-β (Aβ) is unknown, while it plays an important role in the onset and progression of Alzheimer's disease (AD). Aβ polymerization leads to enhanced neurotoxicity, persistent degradation, and deposition in the brain, resulting in decreased Aβ clearance. Diabetes mellitus and poor sleep quality are representative risk factors for the development of AD. Methylglyoxal (MGO) and acrolein are increased in diabetic patients, a representative disease with high glycative stress, and melatonin secretion is decreased during poor sleep quality. In this study, we focused on the Aβ phagocytosis of microglia, which plays a role in Aβ clearance, and examined the effects of Aβ glycation and melatonin. Glycated Aβ was prepared by MGO or acrolein treatment. Fluorescently labeled TAMRA-Aβ and primary rat microglial cells (Cosmo Bio) were used in the experiments. Several new findings were obtained from this experiment. First, microglia phagocytose Aβ, while their phagocytic capacity for glycated Aβ was markedly reduced. Second, Aβ phagocytosis was enhanced by melatonin. Concurrently, spontaneous death of cultured microglia was greater when Aβ was not added than when Aβ was. These findings suggest that prevention of Aβ glycation by countermeasures against glycative stress and prevention of AD progression by lifestyle, <i>i.e.</i>, improvement of sleep quality, are important, rather than elimination of Aβ as has been conventionally practiced.
DOI
10.24659/gsr.9.3_135
Access Restrictions
インターネット公開
Data Provider (Database)
科学技術振興機構 : J-STAGE

Digital

Summary, etc.
The physiological role of amyloid-β (Aβ) is unknown, while it plays an important role in the onset and progression of Alzheimer's disease (AD). Aβ polymerization leads to enhanced neurotoxicity, persistent degradation, and deposition in the brain, resulting in decreased Aβ clearance. Diabetes mellitus and poor sleep quality are representative risk factors for the development of AD. Methylglyoxal (MGO) and acrolein are increased in diabetic patients, a representative disease with high glycative stress, and melatonin secretion is decreased during poor sleep quality. In this study, we focused on the Aβ phagocytosis of microglia, which plays a role in Aβ clearance, and examined the effects of Aβ glycation and melatonin. Glycated Aβ was prepared by MGO or acrolein treatment. Fluorescently labeled TAMRA-Aβ and primary rat microglial cells (Cosmo Bio) were used in the experiments. Several new findings were obtained from this experiment. First, microglia phagocytose Aβ, while their phagocytic capacity for glycated Aβ was markedly reduced. Second, Aβ phagocytosis was enhanced by melatonin. Concurrently, spontaneous death of cultured microglia was greater when Aβ was not added than when Aβ was. These findings suggest that prevention of Aβ glycation by countermeasures against glycative stress and prevention of AD progression by lifestyle, <i>i.e.</i>, improvement of sleep quality, are important, rather than elimination of Aβ as has been conventionally practiced.
Access Restrictions
インターネット公開
Data Provider (Database)
国立情報学研究所 : CiNii Research
Original Data Provider (Database)
Japan Link Center
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