A model of liver carcinogenesis originating from hepatic progenitor cells with accumulation of genetic alterations

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DOI[10.14989/doctor.k18891]to the data of the same series

A model of liver carcinogenesis originating from hepatic progenitor cells with accumulation of genetic alterations

Persistent ID (NDL): info:ndljp/pid/9491586

Material type: 博士論文

Author: Kim, Soo Ki

Publisher: -

Publication date: 2015-03-23

Material Format: Digital

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Name of awarding university/degree: 京都大学,博士(医学),Doctor of Medical Science

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Material Type: 博士論文
Title: A model of liver carcinogenesis originating from hepatic progenitor cells with accumulation of genetic alterations
Author/Editor: Kim, Soo Ki
Author Heading: Kim, Soo Ki
Publication Date: 2015-03-23
Publication Date (W3CDTF): 2015-03-23
Alternative Title: 肝幹/前駆細胞を起源とする肝発癌モデルマウスの確立
Contributor: 松田, 道行
小川, 誠司
野田, 亮
Degree grantor/type: 京都大学
Date Granted: 2015-03-23
Date Granted (W3CDTF): 2015-03-23
Dissertation Number: 甲第18891号
Degree Type: 博士(医学)
Doctor of Medical Science
Conferring No. (Dissertation): 甲第18891号
Text Language Code: eng
Alias of Author: 金, 秀基
キン, スギ
Subject Heading: 肝細胞癌
肝前駆細胞
遺伝子異常
肝発癌
Activation-induced cytidine deaminase (AID)
NDC: 490
Target Audience: 一般
DOI: 10.14989/doctor.k18891
https://doi.org/10.14989/doctor.k18891
Persistent ID (NDL): info:ndljp/pid/9491586
https://dl.ndl.go.jp/pid/9491586
Collection: 障害者向け資料
Collection (Materials For Handicapped People:1): テキストデータ
Collection (particular): 国立国会図書館デジタルコレクション > デジタル化資料 > 博士論文
https://dl.ndl.go.jp/collections/A00014
Acquisition Basis: 博士論文（自動収集）
Date Accepted (W3CDTF): 2015-09-01T13:12:47+09:00
Date Created (W3CDTF): 2015-06-11
Format (IMT): application/pdf
Access Restrictions: 国立国会図書館内限定公開
Service for the Digitized Contents Transmission Service: 図書館・個人送信対象外
Availability of remote photoduplication service: 可
Periodical Title (URI): http://dx.doi.org/10.14989/doctor.k18891
http://hdl.handle.net/2433/199200
Data Provider (Database): 国立国会図書館 : 国立国会図書館デジタルコレクション
https://dl.ndl.go.jp

A model of liver carcinogenesis originating from hepatic progenitor cells with accumulation of genetic alterations

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