Mast Cells Can Amplify Airway Reactivity and Features of Chronic Inflammation in an Asthma Model in Mice
Mast cell–derived TNF contributes to airway hyperreactivity, inflammation, and TH2 cytokine production in an asthma model in mice
Zinc Signals Are Essential for Lipopolysaccharide-Induced Signal Transduction in Monocytes
TNF can contribute to multiple features of ovalbumin-induced allergic inflammation of the airways in mice
Aluminium Hydroxide Adjuvant Initiates Strong Antigen-Specific Th2 Responses in the Absence of IL-4- or IL-13-Mediated Signaling
Eosinophilic Inflammation in Asthma
Visualization of labile zinc and its role in apoptosis of primary airway epithelial cells and cell lines
The role of allergy in the development of asthma
SUBEPITHELIAL FIBROSIS IN THE BRONCHI OF ASTHMATICS
Complexity and Redundancy in the Pathogenesis of Asthma: Reassessing the Roles of Mast Cells and T Cells
Zinc: Mechanisms of Host Defense1,
Protection by the heavy metal chelator N,N,N′,N′-tetrakis (2-Pyridylmethyl)ethylenediamine (TPEN) against the lethal action of botulinum neurotoxin A and B
Modulating the immune response by oral zinc supplementation: a single approach for multiple diseases
Chronic exposure to TNF-α increases airway mucus gene expression
IgE-Dependent Mast Cell Activation Potentiates Airway Responses in Murine Asthma Models
Zinc signals promote IL‐2‐dependent proliferation of T cells
Airway mucus hypersecretion in asthma: an undervalued pathology?
Signalling through the high-affinity IgE receptor FcεRI
Effect of metal chelating agents on the direct and seizure-related neuronal death induced by zinc and kainic acid
The development of allergic inflammation
Zinc metabolism in airway epithelium and airway inflammation: basic mechanisms and clinical targets. A review
Diverse effects of metal chelating agents on the neuronal cytotoxicity of zinc in the hippocampus
Differential Regulation by Glucocorticoid of Interleukin-13–induced Eosinophilia, Hyperresponsiveness, and Goblet Cell Hyperplasia in Mouse Airways
IMMUNOLOGIC BASIS OF ANTIGEN-INDUCED AIRWAY HYPERRESPONSIVENESS
Zinc chelators inhibit eotaxin, RANTES, and MCP-1 production in stimulated human airway epithelium and fibroblasts
A<scp>sthma</scp>: Mechanisms of Disease Persistence and Progression
Functional Significance of Zinc-Related Signaling Pathways in Immune Cells
Zinc Is Required for FcεRI-Mediated Mast Cell Activation
TNF-α induces the late-phase airway hyperresponsiveness and airway inflammation through cytosolic phospholipase A2 activation