Glycative stress and skeletal muscle dysfunctions : as an inducer of "Exercise-Resistance"
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DOI[10.24659/gsr.9.4_199]to the data of the same series
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- Material Type
- 記事
- Author/Editor
- Tatsuro EgawaTakeshi OgawaTakumi Yokokawa
- Publication, Distribution, etc.
- Publication Date
- 2022-12-31
- Publication Date (W3CDTF)
- 2022-12-31
- Alternative Title
- 糖化ストレスによる骨格筋機能低下 : 運動抵抗性誘導因子としての着目
- Periodical title
- Glycative stress research
- No. or year of volume/issue
- 9(4)
- Volume
- 9(4)
- ISSN (Periodical Title)
- 2188-3610
- ISSN-L (Periodical Title)
- 2188-3610
- Text Language Code
- engjpn
- DOI
- 10.24659/gsr.9.4_199
- Persistent ID (NDL)
- info:ndljp/pid/13120789
- Collection
- Collection (Materials For Handicapped People:1)
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Acquisition Basis
- オンライン資料収集制度
- Date Accepted (W3CDTF)
- 2023-12-08T11:19:22+09:00
- Date Captured (W3CDTF)
- 2023-05-20
- Format (IMT)
- application/pdf
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Periodical Title (URI)
- Periodical Title (Persistent ID (NDL))
- info:ndljp/pid/13120786
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館デジタルコレクション
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Holding library
- 国立国会図書館
- Call No.
- Z63-D541
- Related Material (URI)
- Related Material (Persistent ID (NDL))
- info:ndljp/pid/13120789
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館雑誌記事索引
- Bibliographic ID (NDL)
- 032834821
- Bibliographic Record Category (NDL)
- 632
- Summary, etc.
- Skeletal muscle, the largest tissue in the body, is often overlooked for its role as a locomotor organ, however over the past few decades it has been revealed that it also has an important role as a metabolic organ. In recent years, its role as an endocrine organ that controls the homeostatic functions of organs throughout the body mediated by myokine secretion has come under close scrutiny. Skeletal muscle is indispensable for our daily life activities, and in order to maintain its function, it is necessary to understand the factors that deteriorate muscle function and establish a countermeasure. Glycative stress has recently received attention as a factor that impairs skeletal muscle function. Accumulation of advanced glycation end products (AGEs) in skeletal muscle impairs contractile function and myogenic potential. Furthermore, AGEs in the blood elicit inflammatory signals through binding to RAGE (Receptor for AGEs) expressed on muscle cells, resulting in muscle proteolysis. Habitual exercise is important to mitigate the negative effects of such glycative stress on skeletal muscle. On the other hand, it is known that the beneficial effects of exercise vary among individuals. The state in which the effects of exercise are difficult to obtain is called "exercise-resistance," and we hypothesize that glycative stress may be one of the causes of exercise-resistance. In this paper, we will discuss the possibility of glycative stress as an inducer of exercise resistance and summarize its impacts on skeletal muscle.
- DOI
- 10.24659/gsr.9.4_199
- Access Restrictions
- インターネット公開
- Data Provider (Database)
- 科学技術振興機構 : J-STAGE
- Summary, etc.
- Skeletal muscle, the largest tissue in the body, is often overlooked for its role as a locomotor organ, however over the past few decades it has been revealed that it also has an important role as a metabolic organ. In recent years, its role as an endocrine organ that controls the homeostatic functions of organs throughout the body mediated by myokine secretion has come under close scrutiny. Skeletal muscle is indispensable for our daily life activities, and in order to maintain its function, it is necessary to understand the factors that deteriorate muscle function and establish a countermeasure. Glycative stress has recently received attention as a factor that impairs skeletal muscle function. Accumulation of advanced glycation end products (AGEs) in skeletal muscle impairs contractile function and myogenic potential. Furthermore, AGEs in the blood elicit inflammatory signals through binding to RAGE (Receptor for AGEs) expressed on muscle cells, resulting in muscle proteolysis. Habitual exercise is important to mitigate the negative effects of such glycative stress on skeletal muscle. On the other hand, it is known that the beneficial effects of exercise vary among individuals. The state in which the effects of exercise are difficult to obtain is called "exercise-resistance," and we hypothesize that glycative stress may be one of the causes of exercise-resistance. In this paper, we will discuss the possibility of glycative stress as an inducer of exercise resistance and summarize its impacts on skeletal muscle.
- DOI
- 10.24659/gsr.9.4_199
- Access Restrictions
- インターネット公開
- Related Material (URI)
- Data Provider (Database)
- 国立情報学研究所 : CiNii Research
- Original Data Provider (Database)
- Japan Link Center学術機関リポジトリデータベース雑誌記事索引データベース雑誌記事索引データベース科学研究費助成事業データベース科学研究費助成事業データベース
- Bibliographic ID (NDL)
- 03283482113120789