Antiglycative effect of plant extract complex
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DOI[10.24659/gsr.10.1_6]to the data of the same series
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- Material Type
- 記事
- Author/Editor
- Masayuki YagiChieko SakiyamaKyoko Mori
- Publication, Distribution, etc.
- Publication Date
- 2023-03-31
- Publication Date (W3CDTF)
- 2023-03-31
- Alternative Title
- 植物抽出液コンプレックスの抗糖化作用
- Periodical title
- Glycative stress research
- No. or year of volume/issue
- 10(1)
- Volume
- 10(1)
- ISSN (Periodical Title)
- 2188-3610
- ISSN-L (Periodical Title)
- 2188-3610
- Text Language Code
- engjpn
- DOI
- 10.24659/gsr.10.1_6
- Persistent ID (NDL)
- info:ndljp/pid/13120794
- Collection
- Collection (Materials For Handicapped People:1)
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Acquisition Basis
- オンライン資料収集制度
- Date Accepted (W3CDTF)
- 2023-12-08T11:19:22+09:00
- Date Captured (W3CDTF)
- 2023-05-20
- Format (IMT)
- application/pdf
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Periodical Title (URI)
- Periodical Title (Persistent ID (NDL))
- info:ndljp/pid/13120792
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館デジタルコレクション
- Collection (particular)
- 国立国会図書館デジタルコレクション > 電子書籍・電子雑誌 > その他
- Access Restrictions
- 国立国会図書館内限定公開
- Service for the Digitized Contents Transmission Service
- 図書館・個人送信対象外
- Availability of remote photoduplication service
- 可
- Holding library
- 国立国会図書館
- Call No.
- Z63-D541
- Related Material (URI)
- Related Material (Persistent ID (NDL))
- info:ndljp/pid/13120794
- Data Provider (Database)
- 国立国会図書館 : 国立国会図書館雑誌記事索引
- Bibliographic ID (NDL)
- 033430108
- Bibliographic Record Category (NDL)
- 632
- Summary, etc.
- Accumulation of advanced glycation end products (AGEs) in skin tissues due to glycative stress is one of the factors that accelerate skin aging, including decreased elasticity and loss of texture. Suppression of glycative stress is called anti-glycation or glycation care. For anti-glycation of the skin, there are treatments from the inside of the body such as diet and approaches from the outside of the body such as skin care preparations. Furthermore, anti-glycation includes the suppression of postprandial hyperglycemia, suppression of glycation reactions, and decomposition and excretion of AGEs. Various plant materials have been shown to have anti-glycation effects. However, the glycation reaction in the body that leads to the formation of AGEs is a complex multi-pathway reaction, and it is thought that a multi-component approach may be useful. This study was conducted to evaluate the anti-glycation effects of five plant extract complexes (sea buckthorn fruit, Chinese blackberry tea, tea plant leaves, loquat leaves, and rosemary leaves) that can be used in skin care formulations. To evaluate the anti-glycation effects of plant extract complex (PEC) on skin, we examined its inhibitory effects on glycation reaction, AGE cross-link cleavage, inhibitory formation of glycated protein cross-link, glycated protein cross-link cleavage, and antioxidant activity. As results, PEC inhibited the production of fluorescent AGEs in protein glycation models of human serum albumin (HSA), collagen, and keratin. PEC also inhibited the production of pentosidine, <i>N<sup>ε</sup></i>-(carboxymethyl)lysine (CML), 3-deoxyglucosone (3DG), and glyoxal (GO) in a keratin-glucose glycation model. Furthermore, PEC has AGE cross-link-cleavage effect, inhibits protein cross-link formation in a lysozyme-glucose glycation cross-linking model, and degrades proteins dimerized by glycation. PECs may be useful in preventing glycation of skin by suppressing AGE accumulation in skin proteins and preventing functional deterioration caused by the formation of glycated protein cross-links.
- DOI
- 10.24659/gsr.10.1_6
- Access Restrictions
- インターネット公開
- Data Provider (Database)
- 科学技術振興機構 : J-STAGE
- Summary, etc.
- Accumulation of advanced glycation end products (AGEs) in skin tissues due to glycative stress is one of the factors that accelerate skin aging, including decreased elasticity and loss of texture. Suppression of glycative stress is called anti-glycation or glycation care. For anti-glycation of the skin, there are treatments from the inside of the body such as diet and approaches from the outside of the body such as skin care preparations. Furthermore, anti-glycation includes the suppression of postprandial hyperglycemia, suppression of glycation reactions, and decomposition and excretion of AGEs. Various plant materials have been shown to have anti-glycation effects. However, the glycation reaction in the body that leads to the formation of AGEs is a complex multi-pathway reaction, and it is thought that a multi-component approach may be useful. This study was conducted to evaluate the anti-glycation effects of five plant extract complexes (sea buckthorn fruit, Chinese blackberry tea, tea plant leaves, loquat leaves, and rosemary leaves) that can be used in skin care formulations. To evaluate the anti-glycation effects of plant extract complex (PEC) on skin, we examined its inhibitory effects on glycation reaction, AGE cross-link cleavage, inhibitory formation of glycated protein cross-link, glycated protein cross-link cleavage, and antioxidant activity. As results, PEC inhibited the production of fluorescent AGEs in protein glycation models of human serum albumin (HSA), collagen, and keratin. PEC also inhibited the production of pentosidine, <i>N<sup>ε</sup></i>-(carboxymethyl)lysine (CML), 3-deoxyglucosone (3DG), and glyoxal (GO) in a keratin-glucose glycation model. Furthermore, PEC has AGE cross-link-cleavage effect, inhibits protein cross-link formation in a lysozyme-glucose glycation cross-linking model, and degrades proteins dimerized by glycation. PECs may be useful in preventing glycation of skin by suppressing AGE accumulation in skin proteins and preventing functional deterioration caused by the formation of glycated protein cross-links.
- DOI
- 10.24659/gsr.10.1_6
- Related Material (URI)
- Data Provider (Database)
- 国立情報学研究所 : CiNii Research
- Original Data Provider (Database)
- Japan Link Center雑誌記事索引データベース雑誌記事索引データベース
- Bibliographic ID (NDL)
- 03343010813120794